PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8814547-5	1996	Treatment with organic hydrogen peroxides mimicked UVB radiation in upregulating c-jun expression, suggesting the participation of reactive oxygen intermediates in the UVB-signaling pathway.	reactive oxygen	131-146	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	81-86	
12682616-17	2003	Reactive oxygen intermediates can also function as signaling messengers to activate transcription factors, like NFkB and AP-1, and induce gene expression.	reactive oxygen	0-15	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	121-125	
7724734-2	1995	In these cell types, the radiation- and H2O2-mediated increase in c-jun mRNA levels could be prevented by pretreatment of the cells with N-acetylcysteine, an antioxidant, or H7, an inhibitor of protein kinase C and protein kinase A, but not by HA1004, a specific inhibitor of protein kinase A and G. These results suggest a role for protein kinase C and reactive oxygen intermediates in the induction of c-jun gene expression in both normal and tumor cells.	reactive oxygen	354-369	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	66-71