PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27677293-0	2017	A Quinone-Containing Compound Enhances Camptothecin-Induced Apoptosis of Lung Cancer Through Modulating Endogenous ROS and ERK Signaling.	quinone	2-9	mitogen-activated protein kinase 1	Homo sapiens	123-126	
15795063-0	2005	Benzoquinone activates the ERK/MAPK signaling pathway via ROS production in HL-60 cells.	quinone	0-12	mitogen-activated protein kinase 1	Homo sapiens	27-30	
15795063-3	2005	Therefore, we explored the mechanisms underlying BQ-induced HL-60 cell proliferation by studying the role of BQ-induced reactive oxygen species (ROS) in the activation of the ERK-MAPK signaling pathway.	quinone	109-111	mitogen-activated protein kinase 1	Homo sapiens	175-178	
15652497-0	2005	Quinone-induced Cdc25A inhibition causes ERK-dependent connexin phosphorylation.	quinone	0-7	mitogen-activated protein kinase 1	Homo sapiens	41-44	
15795063-9	2005	Simultaneous treatment with catalase or PD98059, a potent MEK protein inhibitor, reduced cell recruitment into the S-phase and also abolished the ERK1/2 protein phosphorylation induced by BQ, suggesting that MEK/ERK is an important pathway involved in BQ-induced ROS mediated proliferation.	quinone	188-190	mitogen-activated protein kinase 1	Homo sapiens	146-149