PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26883384-3	2016	Here we showed that deletion of ACE2 aggravates liver steatosis, which is correlated with the increased expression of hepatic lipogenic genes and the decreased expression of fatty acid oxidation-related genes in the liver of ACE2 knockout (ACE2(-/y)) mice.	Fatty Acids	174-184	angiotensin I converting enzyme (peptidyl-dipeptidase A) 2	Mus musculus	32-36	
26883384-3	2016	Here we showed that deletion of ACE2 aggravates liver steatosis, which is correlated with the increased expression of hepatic lipogenic genes and the decreased expression of fatty acid oxidation-related genes in the liver of ACE2 knockout (ACE2(-/y)) mice.	Fatty Acids	174-184	angiotensin I converting enzyme (peptidyl-dipeptidase A) 2	Mus musculus	225-229	
26883384-3	2016	Here we showed that deletion of ACE2 aggravates liver steatosis, which is correlated with the increased expression of hepatic lipogenic genes and the decreased expression of fatty acid oxidation-related genes in the liver of ACE2 knockout (ACE2(-/y)) mice.	Fatty Acids	174-184	angiotensin I converting enzyme (peptidyl-dipeptidase A) 2	Mus musculus	225-229	
26883384-5	2016	On the contrary, overexpression of ACE2 improved fatty liver in db/db mice, and the mRNA levels of fatty acid oxidation-related genes were up-regulated.	Fatty Acids	99-109	angiotensin I converting enzyme (peptidyl-dipeptidase A) 2	Mus musculus	35-39