PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12865424-4	2003	Saturated fatty acid (lauric acid)-induced NFkappaB activation was inhibited by a dominant-negative mutant of TLR4, MyD88, IRAK-1, TRAF6, or IkappaBalpha in macrophages (RAW264.7) and 293T cells transfected with TLR4 and MD2.	Fatty Acids	0-20	myeloid differentiation primary response gene 88	Mus musculus	116-121	
34290146-4	2021	We show here that short fatty acid (FA) chain sulfatides directly activate mouse TLR4-MD-2 independent of CD14, trigger MyD88- and TRIF-dependent signaling, and stimulate tumor necrosis factor alpha (TNFalpha) and type I interferon (IFN) production in mouse macrophages.	Fatty Acids	24-34	myeloid differentiation primary response gene 88	Mus musculus	120-125	
31427815-6	2019	De novo fatty acid synthesis and CD36-mediated exogenous fatty acid incorporation contributed to MYD88 palmitoylation.	Fatty Acids	8-18	myeloid differentiation primary response gene 88	Mus musculus	97-102	
31427815-6	2019	De novo fatty acid synthesis and CD36-mediated exogenous fatty acid incorporation contributed to MYD88 palmitoylation.	Fatty Acids	57-67	myeloid differentiation primary response gene 88	Mus musculus	97-102	
31427815-9	2019	Thus, intracellular saturated fatty acid-dependent palmitoylation of MYD88 by ZDHHC6 is a therapeutic target of sepsis.	Fatty Acids	20-40	myeloid differentiation primary response gene 88	Mus musculus	69-74	
19808018-0	2009	MyD88 signaling in the CNS is required for development of fatty acid-induced leptin resistance and diet-induced obesity.	Fatty Acids	58-68	myeloid differentiation primary response gene 88	Mus musculus	0-5