PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28633967-9	2017	We have also shown that FAAH inhibition improved morphology of coronary blood vessels and only partially maintained its effect on lipid metabolism in the DOCA-salt hearts (i.e. elevated plasma and intramyocardial TAG contents as well as plasmalemmal FAT/CD36 and total FATP1 expressions).	Desoxycorticosterone Acetate	154-158	fatty-acid amide hydrolase-like	Rattus norvegicus	24-28	
26969765-10	2016	Reduction in BP after chronic administration of the FAAH inhibitor URB597 in DOCA-salt hypertensive rats only partially correlates with structural and functional changes in conductance and resistance vessels, respectively.	Desoxycorticosterone Acetate	77-81	fatty-acid amide hydrolase-like	Rattus norvegicus	52-56	
27086176-3	2016	The aim of this study was to evaluate the effects of chronic administration of the fatty acid amide hydrolase (FAAH) inhibitor [3-(3-carbamoylphenyl)phenyl]N-cyclohexylcarbamate (URB597) on the endocannabinoid system and on the redox balance in the livers of DOCA-salt hypertensive rats.	Desoxycorticosterone Acetate	259-263	fatty-acid amide hydrolase-like	Rattus norvegicus	111-115	
26969765-1	2016	AIMS: This study examined whether the fall in blood pressure (BP) induced by the chronic inhibition of fatty acid amide hydrolase (FAAH) by URB597 in deoxycorticosterone acetate (DOCA-salt) hypertensive rats correlates with endocannabinoid-mediated vascular changes.	Desoxycorticosterone Acetate	150-177	fatty-acid amide hydrolase-like	Rattus norvegicus	131-135	
29197803-8	2018	The administration of FAAH inhibitor resulted in increased level of endocannabinoids in kidney of both groups of hypertensive rats led to enhanced expression of the cannabinoid receptors type 1 and 2 in SHR as well as vanilloid receptor 1 receptors in DOCA-salt rats.	Desoxycorticosterone Acetate	252-256	fatty-acid amide hydrolase-like	Rattus norvegicus	22-26