PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14694538-10	2004	Resiniferatoxin reduced CYP-induced up-regulation of p-CREB in DRG, suggesting that cystitis can reveal an altered CREB phosphorylation that may be mediated by capsaicin-sensitive bladder afferents.	Capsaicin	160-169	cAMP responsive element binding protein 1	Homo sapiens	115-119	
27979859-9	2017	Capsaicin induced phosphorylation of CREB by raising [Ca2+]cyt, and capsaicin-induced CREB phosphorylation were significantly enhanced in IPAH PASMCs compared with normal PASMCs.	Capsaicin	0-9	cAMP responsive element binding protein 1	Homo sapiens	37-41	
27979859-9	2017	Capsaicin induced phosphorylation of CREB by raising [Ca2+]cyt, and capsaicin-induced CREB phosphorylation were significantly enhanced in IPAH PASMCs compared with normal PASMCs.	Capsaicin	68-77	cAMP responsive element binding protein 1	Homo sapiens	86-90	
18519133-5	2008	The effect of capsaicin was long lasting and the neurons were found to be hyperfunctional and with increased levels of phosphorylated CREB (cAMP responsive element binding) even after 72 h of treatment with capsaicin (32+/-5% vs. 52+/-5%).	Capsaicin	14-23	cAMP responsive element binding protein 1	Homo sapiens	134-138	
18519133-5	2008	The effect of capsaicin was long lasting and the neurons were found to be hyperfunctional and with increased levels of phosphorylated CREB (cAMP responsive element binding) even after 72 h of treatment with capsaicin (32+/-5% vs. 52+/-5%).	Capsaicin	14-23	cAMP responsive element binding protein 1	Homo sapiens	140-171	
18519133-5	2008	The effect of capsaicin was long lasting and the neurons were found to be hyperfunctional and with increased levels of phosphorylated CREB (cAMP responsive element binding) even after 72 h of treatment with capsaicin (32+/-5% vs. 52+/-5%).	Capsaicin	207-216	cAMP responsive element binding protein 1	Homo sapiens	134-138	
18519133-5	2008	The effect of capsaicin was long lasting and the neurons were found to be hyperfunctional and with increased levels of phosphorylated CREB (cAMP responsive element binding) even after 72 h of treatment with capsaicin (32+/-5% vs. 52+/-5%).	Capsaicin	207-216	cAMP responsive element binding protein 1	Homo sapiens	140-171	
18519133-7	2008	The results suggest the participation of Ca2+, CaMKII and CREB on the prolonged enhancement of excitability following chronic exposure to capsaicin.	Capsaicin	138-147	cAMP responsive element binding protein 1	Homo sapiens	58-62	
14622772-0	2002	The role of nitric oxide in the phosphorylation of cyclic adenosine monophosphate-responsive element-binding protein in the spinal cord after intradermal injection of capsaicin.	Capsaicin	167-176	cAMP responsive element binding protein 1	Homo sapiens	51-116	
14622772-3	2002	The level of p-CREB increased by 20 minutes, peaked between 20 and 60 minutes after capsaicin injection, and started to decrease after 150 minutes.	Capsaicin	84-93	cAMP responsive element binding protein 1	Homo sapiens	15-19	
14622772-5	2002	The p-CREB expression on the ipsilateral side of the spinal dorsal horn, but not on the contralateral side, increased significantly after capsaicin injection.	Capsaicin	138-147	cAMP responsive element binding protein 1	Homo sapiens	6-10	
14622772-6	2002	The increase in p-CREB induced by capsaicin injection was partially blocked by pretreatment with N(G)-nitro-L-arginine methyl ester (L-NAME), an NO synthase inhibitor, administered through a microdialysis fiber placed across the spinal cord.	Capsaicin	34-43	cAMP responsive element binding protein 1	Homo sapiens	18-22