PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10202860-0 1999 Central injections of capsaicin cause antidiuresis mediated through neurokinin-1 receptors in rat hypothalamus and vasopressin release. Capsaicin 22-31 arginine vasopressin Rattus norvegicus 115-126 16196077-7 2006 When examining potential candidates for the mediation of capsaicin-induced constriction, we found that vasopressin (VP), NPY, serotonin (5HT) and endothelin (ET), but not neurokinin A or substance P, caused a concentration-dependent vasoconstriction of epineurial arterioles. Capsaicin 57-66 arginine vasopressin Rattus norvegicus 103-114 16196077-7 2006 When examining potential candidates for the mediation of capsaicin-induced constriction, we found that vasopressin (VP), NPY, serotonin (5HT) and endothelin (ET), but not neurokinin A or substance P, caused a concentration-dependent vasoconstriction of epineurial arterioles. Capsaicin 57-66 arginine vasopressin Rattus norvegicus 116-118 10202860-5 1999 These findings suggested that capsaicin stimulated substance P release in the SON and caused the antidiuresis as a result of the increased release of vasopressin into the circulation from the neurohypophysis mediated through neurokinin-1 receptors in the SON. Capsaicin 30-39 arginine vasopressin Rattus norvegicus 150-161 3429461-0 1987 Capsaicin-induced inhibition of motility of the rat isolated vas deferens: do multiple neuropeptides mediate the visceromotor effects of capsaicin? Capsaicin 0-9 arginine vasopressin Rattus norvegicus 61-64 2690648-0 1989 Synthetic capsaicin reversibly impairs vasopressin-mediated blood pressure recovery. Capsaicin 10-19 arginine vasopressin Rattus norvegicus 39-50 2690648-1 1989 The vasopressin-mediated recovery of arterial pressure observed in adult rats following pharmacological blockade of the sympathetic nervous and renin-angiotensin systems is reduced by neonatal capsaicin treatment. Capsaicin 193-202 arginine vasopressin Rattus norvegicus 4-15 7712013-2 1995 Capsaicin, a selective sensory neurotoxin, was given to newborn rats and at the age of 3 months the contractile activity of the urinary bladder detrusor muscle and vas deferens evoked by either electrical field stimulation (EFS) or exogenous adenosine 5"-triphosphate (ATP) and carbachol (urinary bladder), or ATP and noradrenaline (vas deferens) were tested. Capsaicin 0-9 arginine vasopressin Rattus norvegicus 333-336 7712013-13 1995 Contractions of the vas deferens evoked by either ATP (10 micro M-3 mM) or noradrenaline (1-1000 micro M) in the capsaicin-treated group showed no significant difference between control and capsaicin treated rats.6. Capsaicin 113-122 arginine vasopressin Rattus norvegicus 20-23 3429461-10 1987 5 The inhibitory effect of capsaicin or CGRP on the Kassinin-stimulated rhythmic contractions of the rat isolated vas deferens was unaffected by a previous exposure to tetrodotoxin (0.5 microM). Capsaicin 27-36 arginine vasopressin Rattus norvegicus 114-117 3429461-14 1987 7 These findings indicate the existence, in the rat isolated vas deferens, of capsaicin-sensitive sensory innervation which, upon chemical stimulation, releases, through a tetrodotoxin-insensitive mechanism, a substance(s) which inhibits motility at postjunctional level. Capsaicin 78-87 arginine vasopressin Rattus norvegicus 61-64 3497262-2 1987 Capsaicin attenuated the twitch response of the rat vas deferens induced by the transmural nerve stimulation (TNS) in vitro. Capsaicin 0-9 arginine vasopressin Rattus norvegicus 52-55 3497262-11 1987 These results suggest that capsaicin releases endogenous CGRP and that the released CGRP inhibits the contraction of the rat vas deferens by acting directly on smooth muscle cells but not on the sympathetic nerves. Capsaicin 27-36 arginine vasopressin Rattus norvegicus 125-128 3899238-4 1985 During angiotensin converting enzyme inhibition (with captopril) and ganglion blockade (with pentolinium), the vasopressin-mediated blood pressure recovery was more gradual in the capsaicin-treated animals than in the controls, but after 60 min blood pressures were similar in all groups. Capsaicin 180-189 arginine vasopressin Rattus norvegicus 111-122 3039423-0 1987 Immunoblockade of response to capsaicin in the rat vas deferens: evidence for the involvement of endogenous calcitonin gene-related peptide. Capsaicin 30-39 arginine vasopressin Rattus norvegicus 51-54 3039423-1 1987 In the rat isolated vas deferens, capsaicin induced a transitory inhibition of the nerve-mediated contractions. Capsaicin 34-43 arginine vasopressin Rattus norvegicus 20-23 2426633-5 1986 Arginine vasopressin-like immunoreactivity was present also in laminae I and II of the dorsal horn of the spinal cord and this reactivity was absent in animals which had been treated neonatally with capsaicin, suggesting that it was contained in primary afferent terminals. Capsaicin 199-208 arginine vasopressin Rattus norvegicus 9-20 3513052-3 1986 Furthermore, following administration of captopril and pentolinium, the capsaicin-treated animals showed marked impairment of the vasopressin-dependent recovery of blood pressure. Capsaicin 72-81 arginine vasopressin Rattus norvegicus 130-141 3513052-5 1986 Long-Evans rats treated neonatally with capsaicin may be due to less effective compensation for inhibition of the renin-angiotensin system when vasopressin release is impaired. Capsaicin 40-49 arginine vasopressin Rattus norvegicus 144-155 3899238-5 1985 Collectively the results indicate that although the full development of vasopressin-dependent mechanisms following acute hypotension takes longer when a large proportion of unmyelinated afferent fibres have been destroyed by neonatal treatment with capsaicin, 48 h of water deprivation results in a normal involvement of vasopressin-dependent mechanisms in the maintenance of blood pressure. Capsaicin 249-258 arginine vasopressin Rattus norvegicus 72-83 3899238-5 1985 Collectively the results indicate that although the full development of vasopressin-dependent mechanisms following acute hypotension takes longer when a large proportion of unmyelinated afferent fibres have been destroyed by neonatal treatment with capsaicin, 48 h of water deprivation results in a normal involvement of vasopressin-dependent mechanisms in the maintenance of blood pressure. Capsaicin 249-258 arginine vasopressin Rattus norvegicus 321-332 3896362-0 1985 Pressor sensitivity to exogenous vasopressin in conscious, adult rats treated neonatally with capsaicin. Capsaicin 94-103 arginine vasopressin Rattus norvegicus 33-44 3896362-7 1985 From the present findings, it seems that our previous observation of impaired, vasopressin-mediated blood pressure recovery following acute hypotension in capsaicin-treated rats cannot be attributed to a reduced pressor sensitivity to the hormone. Capsaicin 155-164 arginine vasopressin Rattus norvegicus 79-90 6704593-0 1984 Neonatal capsaicin treatment impairs vasopressin-mediated blood pressure recovery following acute hypotension. Capsaicin 9-18 arginine vasopressin Rattus norvegicus 37-48 6704593-7 1984 These results suggest that neonatal treatment with capsaicin impairs vasopressin-mediated recovery of blood pressure following acute hypotension. Capsaicin 51-60 arginine vasopressin Rattus norvegicus 69-80 32685762-2 2020 Hypertonic stimulation modulates glutamatergic inputs to the supraoptic nucleus (SON) magnocellular neurosecretory cells (MNCs) putative vasopressin (VP) neurons through capsaicin-insensitive transient receptor potential vanilloid (TRPV) 1 channels on the presynaptic terminals. Capsaicin 170-179 arginine vasopressin Rattus norvegicus 137-148