PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19386012-0 2009 Beta-carotene is incorporated or mobilized along with triglycerides in bovine adipose tissue in response to insulin or epinephrine. Triglycerides 54-67 insulin Bos taurus 108-115 22459825-1 2012 We examined the association between plasma insulin concentration and fatty acid distribution to milk fat and membrane fractions (triglyceride and phospholipid, respectively) in dairy cows. Triglycerides 129-141 insulin Bos taurus 43-50 22459825-5 2012 Fatty acid analysis of milk samples demonstrated a possible association between plasma insulin concentration and the fatty acid composition in phospholipid and triglyceride fractions. Triglycerides 160-172 insulin Bos taurus 87-94 22459825-11 2012 Furthermore, a lower triglyceride-to-phospholipid ratio in the HI group implies that high plasma insulin and low milk fat concentrations are associated with a shift in the size distribution of milk fat globules. Triglycerides 21-33 insulin Bos taurus 97-104 19386012-8 2009 Explants from six animals were incubated with either hormone and 0 or 20 microm beta-C for 20 h. Both TG and beta-C contents were affected positively by insulin and negatively by epinephrine. Triglycerides 102-104 insulin Bos taurus 153-160 9565877-0 1998 Relationship of triglyceride accumulation to insulin clearance and hormonal responsiveness in bovine hepatocytes. Triglycerides 16-28 insulin Bos taurus 45-52 16840615-11 2006 Insulin response was negatively correlated with plasma NEFA and liver triglycerides. Triglycerides 70-83 insulin Bos taurus 0-7 10219522-1 1999 Whole-body insulin-dependent glucose utilization and insulin responses to glucose in euglycemic-hyperinsulinemic clamps (EHGC) and in hyperglycemic clamps (HGC) were evaluated in high yielding dairy cows fed rumen-protected fat (triglycerides), free fatty acids, or a starch-rich ration (n = 5 per group) in Week 9 and Week 19 of lactation. Triglycerides 229-242 insulin Bos taurus 11-18 17517713-16 2007 The induction of insulin resistance by TG, NEFA, or both may increase the availability of glucogenic nutrients to the periparturient dairy cow. Triglycerides 39-41 insulin Bos taurus 17-24 12362450-1 2002 The objective of this experiment was to determine whether there is a dose of slow-release insulin (SRI) that decreases concentrations of plasma nonesterified fatty acids (NEFA) and liver triglyceride (TG) without decreasing plasma glucose concentration, dry matter intake (DMI), and milk yield. Triglycerides 187-199 insulin Bos taurus 90-97 12362450-1 2002 The objective of this experiment was to determine whether there is a dose of slow-release insulin (SRI) that decreases concentrations of plasma nonesterified fatty acids (NEFA) and liver triglyceride (TG) without decreasing plasma glucose concentration, dry matter intake (DMI), and milk yield. Triglycerides 201-203 insulin Bos taurus 90-97 9565877-6 1998 Insulin clearance rates were also lower in hepatocytes loaded with triglycerides than in normal hepatocytes. Triglycerides 67-80 insulin Bos taurus 0-7 9149959-5 1997 Insulin reduced labeled triglyceride depletion by 49%. Triglycerides 24-36 insulin Bos taurus 0-7 9149959-7 1997 Lipid infiltration influenced hepatic function, and insulin regulated hepatic triglyceride concentration. Triglycerides 78-90 insulin Bos taurus 52-59 3902350-1 1985 The effect of insulin on net metabolism of acetate, beta-hydroxybutyrate, glucose and triglycerides by the bovine mammary gland was determined using the glucose clamp technique. Triglycerides 86-99 insulin Bos taurus 14-21 8227621-9 1993 Prepartum plasma glucose, NEFA, BHBA, and insulin were strongly correlated with liver triglyceride at 1 d postpartum (r = -.49, .45, .36, and -.49, respectively). Triglycerides 86-98 insulin Bos taurus 42-49 9101563-6 1997 Insulin injections also increased the amount of abdominal fat, plasma triglycerides, and insulinemia and decreased blood glucose (all p < 0.05). Triglycerides 70-83 insulin Bos taurus 0-7 8456114-8 1993 Hepatocytes incubated in supplemented medium containing bovine insulin had lesser rates of TG synthesis than cells incubated with similar concentrations of chicken insulin. Triglycerides 91-93 insulin Bos taurus 63-70 3902350-0 1985 Effects of insulin on the metabolism of acetate, beta-hydroxybutyrate and triglycerides by the bovine mammary gland. Triglycerides 74-87 insulin Bos taurus 11-18 6760301-8 1982 Blood triglycerides exhibited a small increase at 0.5 h after the meal but a large decrease at 1-4.5 h. The meal also resulted in a very large increase in blood glucose with maximal values occurring at 1-4.5 h. Blood insulin showed a large increment at 0.5 h then increased slowly, peaking at 2-3 h. The postprandial increase in blood insulin was less during the first experimental period than during the second one. Triglycerides 6-19 insulin Bos taurus 217-224 935092-2 1976 A study was made of the effect of actic extract from the epiphysis of cattle on the level of immunoreactive insulin under conditions of glucose load, triglyceride content and insulin tolerance. Triglycerides 150-162 insulin Bos taurus 108-115 5071176-5 1972 Raised concentrations of insulin or addition of fructose increased secretion of triglyceride in very-low-density lipoproteins, but only insulin caused more of the free fatty acids taken up by the liver to be incorporated into very-low-density lipoproteins. Triglycerides 80-92 insulin Bos taurus 25-32 33714908-3 2021 Based on this, the aim of this study was to evaluate the hepatic expression of proteins of the insulin signaling pathway (PI3K) and of the cytokines TNFalpha, IL-6 and NF-kappaB, as well as the plasma concentrations of non-esterified fatty acids (NEFA), beta-hydroxybutyrate, glucose, triglycerides (TAG), insulin and insulin-like growth factor-1, insulin sensitivity indexes, and the hepatic content of TAG during the transition period in cows with different BCS. Triglycerides 285-298 insulin Bos taurus 95-102 29785900-8 2018 Consequently, triglyceride content was significantly increased in insulin treated hepatocytes. Triglycerides 14-26 insulin Bos taurus 66-73 29785900-9 2018 Activation of AMPKalpha induced by AICAR could reverse the effect of insulin on PPARalpha, SREBP-1c, and ChREBP, thereby decreasing triglyceride content. Triglycerides 132-144 insulin Bos taurus 69-76