PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28282785-0	2017	Peimine inhibits hERG potassium channels through the channel inactivation states.	verticine	0-7	ETS transcription factor ERG	Homo sapiens	17-21	
30956677-2	2019	Pharmacological researches have reported various benefits of verticine, including anticancer, anti-inflammatory, protecting against acute lung injury, tracheobronchial relaxation, antitussive, expectorant, sedative, and analgesic activities, in addition to inhibiting proliferation of cultured orbital fibroblast, angiotensin converting enzyme (ACE), and acetylcholinesterase (AChE) and inhibiting hERG potassium channels.	verticine	61-70	ETS transcription factor ERG	Homo sapiens	398-402	
28282785-8	2017	Thus, Peimine was chosen to investigate its inhibitory effects on hERG channels.	verticine	6-13	ETS transcription factor ERG	Homo sapiens	66-70	
28282785-10	2017	RESULTS AND CONCLUSION: We found that Peimine inhibited the hERG peak tail currents in a concentration dependent manner with an IC50 value of 43.7muM (n=4) by whole cell patch clamp techniques.	verticine	38-45	ETS transcription factor ERG	Homo sapiens	60-64	
28282785-16	2017	Mutation of Y652 to Alanine reduced sensitivity to Peimine, suggesting that Y652 is an important hERG binding sites for Peimine.	verticine	51-58	ETS transcription factor ERG	Homo sapiens	97-101	
28282785-16	2017	Mutation of Y652 to Alanine reduced sensitivity to Peimine, suggesting that Y652 is an important hERG binding sites for Peimine.	verticine	120-127	ETS transcription factor ERG	Homo sapiens	97-101