PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15473983-0	2004	Enhanced release of synaptic glutamate underlies the potentiation of oxygen-glucose deprivation-induced neuronal injury after induction of NOS-2.	oxygen-glucose	69-83	nitric oxide synthase 2, inducible	Mus musculus	139-144	
15473983-2	2004	Consistent with this, induction of nitric oxide synthase (NOS-2) in murine neocortical cell cultures potentiated neuronal cell death caused by combined oxygen-glucose deprivation in association with a net increase in extracellular glutamate accumulation.	oxygen-glucose	152-166	nitric oxide synthase 2, inducible	Mus musculus	58-63	
31426811-7	2019	RESULTS: After oxygen-glucose deprivation (OGD), iNOS mRNA expression increased to the peak at 6 h in primary astrocytes, but keep baseline expression in LCN2-knockout astrocytes.	oxygen-glucose	15-29	nitric oxide synthase 2, inducible	Mus musculus	49-53	
8784134-0	1996	Potentiation of oxygen-glucose deprivation-induced neuronal death after induction of iNOS.	oxygen-glucose	16-30	nitric oxide synthase 2, inducible	Mus musculus	85-89	
8784134-8	1996	However, after induction of astrocyte iNOS activity with interferon gamma plus lipopolysaccharide or interleukin-1 beta, oxygen-glucose deprivation-induced neuronal injury was markedly enhanced and nitric oxide synthase inhibitors became protective.	oxygen-glucose	121-135	nitric oxide synthase 2, inducible	Mus musculus	38-42