PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20228169-11	2010	In 3T3-L1 adipocytes, long-term ritonavir exposure perturbs FA metabolism by increasing ATGL-mediated partial TAG hydrolysis, thus increasing FA efflux, and leads to compensatory increases in FA reesterification with glycerol and acylglycerols.	Glycerol	217-225	patatin like phospholipase domain containing 2	Homo sapiens	88-92	
23108672-8	2012	Following CGI-58 knockdown or ATGL inhibition with bromoenol lactone, release of both glycerol and FA was significantly lowered.	Glycerol	86-94	patatin like phospholipase domain containing 2	Homo sapiens	30-34	
23066022-3	2012	Here we show that ATGL exhibits a strong preference for the hydrolysis of long-chain fatty acid esters at the sn-2 position of the glycerol backbone.	Glycerol	131-139	patatin like phospholipase domain containing 2	Homo sapiens	18-22	
21969372-4	2011	Silencing of ATGL expression in adipocytes almost completely abolished basal and TNF-alpha-induced glycerol release.	Glycerol	99-107	patatin like phospholipase domain containing 2	Homo sapiens	13-17	
21969372-5	2011	In comparison, the glycerol release under the same conditions was only partially decreased upon reduction in expression of either HSL or the ATGL coactivator CGI-58.	Glycerol	19-27	patatin like phospholipase domain containing 2	Homo sapiens	141-145