PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3401512-1	1988	[3H]Triamcinolone acetonide glucocorticoid receptor complexes from human salivary gland adenocarcinoma cells (HSG cells) were shown to be activated with an accompanying decrease in molecular weight in intact cells, as analyzed by gel filtration, DEAE chromatography, the mini-column method and glycerol gradient centrifugation.	Glycerol	294-302	nuclear receptor subfamily 3 group C member 1	Homo sapiens	28-51	
2494184-3	1989	Glycerol gradient centrifugation was utilized in order to separate the 6 S GR.DNA complex from the 4 S GR and the 3 S DNA fragment.	Glycerol	0-8	nuclear receptor subfamily 3 group C member 1	Homo sapiens	75-77	
2494184-3	1989	Glycerol gradient centrifugation was utilized in order to separate the 6 S GR.DNA complex from the 4 S GR and the 3 S DNA fragment.	Glycerol	0-8	nuclear receptor subfamily 3 group C member 1	Homo sapiens	103-105	
2494184-10	1989	This suggests that a GR multimer, probably a homodimer, is stable during gel filtration chromatography but needs to be stabilized by glutaraldehyde cross-linking or DNA during glycerol gradient centrifugation.	Glycerol	176-184	nuclear receptor subfamily 3 group C member 1	Homo sapiens	21-23	
29266408-4	2018	In mature 3T3-L1 adipocytes, GCR activation using dexamethasone upregulated adipose triglyceride lipase (ATGL) and downregulated phosphoenolpyruvate carboxykinase (PEPCK), resulting in enhanced glycerol release into the medium.	Glycerol	194-202	nuclear receptor subfamily 3 group C member 1	Homo sapiens	29-32	
29266408-10	2018	In conclusion, crosstalk between GCR and PPARgamma is largely synergistic but counter-regulatory in lipogenic genes, of which enhancement prevents excessive glycerol and possibly FFA release by glucocorticoids into the circulation.	Glycerol	157-165	nuclear receptor subfamily 3 group C member 1	Homo sapiens	33-36