PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 24046867-3 2013 To this end, MC5R expression was decreased by small interfering RNA (siRNA), which significantly impaired the alpha-MSH stimulation of lipolysis, as determined by glycerol and nonesterified fatty-acid (NEFA) quantification. Glycerol 163-171 proopiomelanocortin Homo sapiens 110-119 16155875-3 2005 Similar to ACTH, MSH treatment resulted in the release of glycerol into the cell supernatant. Glycerol 58-66 proopiomelanocortin Homo sapiens 17-20 16155875-5 2005 In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases. Glycerol 237-245 proopiomelanocortin Homo sapiens 158-161 16155875-5 2005 In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases. Glycerol 237-245 proopiomelanocortin Homo sapiens 225-228 16155875-5 2005 In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases. Glycerol 237-245 proopiomelanocortin Homo sapiens 225-228 2951244-1 1987 beta-Endorphin stimulates glycerol release from adipose tissue in vitro in the rabbit. Glycerol 26-34 proopiomelanocortin Homo sapiens 0-14 8429757-4 1993 beta-Endorphin induced a significant increase in glycerol release in isolated human fat cells. Glycerol 49-57 proopiomelanocortin Homo sapiens 0-14 32549368-10 2020 Based on the test of ACTH release from semi-solid formulations and evaluation of rheological parameters, it was found that glycerol ointment is the most favourable base for ACTH. Glycerol 123-131 proopiomelanocortin Homo sapiens 21-25 6324870-1 1984 Inhibitors of protein synthesis, cycloheximide and puromycin, blocked ACTH (adrenocorticotropin)-induced increases in phospholipid mass, including phosphatidylinositol, but paradoxically increase 32P-labelling (but not [3H]glycerol-labelling) therein. Glycerol 223-231 proopiomelanocortin Homo sapiens 70-74 6273120-7 1981 Moreover, the increased incorporation of labeled glycerol and palmitate into phospholipids suggests that ACTH and cAMP may stimulate the glycerol-3"-PO4 acyltransferase reaction. Glycerol 49-57 proopiomelanocortin Homo sapiens 105-109 32549368-10 2020 Based on the test of ACTH release from semi-solid formulations and evaluation of rheological parameters, it was found that glycerol ointment is the most favourable base for ACTH. Glycerol 123-131 proopiomelanocortin Homo sapiens 173-177