PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24046867-3	2013	To this end, MC5R expression was decreased by small interfering RNA (siRNA), which significantly impaired the alpha-MSH stimulation of lipolysis, as determined by glycerol and nonesterified fatty-acid (NEFA) quantification.	Glycerol	163-171	proopiomelanocortin	Homo sapiens	110-119	
32549368-10	2020	Based on the test of ACTH release from semi-solid formulations and evaluation of rheological parameters, it was found that glycerol ointment is the most favourable base for ACTH.	Glycerol	123-131	proopiomelanocortin	Homo sapiens	21-25	
16155875-5	2005	In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases.	Glycerol	237-245	proopiomelanocortin	Homo sapiens	225-228	
8429757-4	1993	beta-Endorphin induced a significant increase in glycerol release in isolated human fat cells.	Glycerol	49-57	proopiomelanocortin	Homo sapiens	0-14	
6324870-1	1984	Inhibitors of protein synthesis, cycloheximide and puromycin, blocked ACTH (adrenocorticotropin)-induced increases in phospholipid mass, including phosphatidylinositol, but paradoxically increase 32P-labelling (but not [3H]glycerol-labelling) therein.	Glycerol	223-231	proopiomelanocortin	Homo sapiens	70-74	
16155875-3	2005	Similar to ACTH, MSH treatment resulted in the release of glycerol into the cell supernatant.	Glycerol	58-66	proopiomelanocortin	Homo sapiens	17-20	
16155875-5	2005	In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases.	Glycerol	237-245	proopiomelanocortin	Homo sapiens	158-161	
16155875-5	2005	In addition, a variety of kinases, including protein kinase A (PKA) and extracellular signal-regulated kinase (ERK) were also phosphorylated as the result of MSH treatment, and their specific inhibitors caused a reduction in MSH-induced glycerol release and HSL activity, indicating that MSH-induced lipolysis was mediated by these kinases.	Glycerol	237-245	proopiomelanocortin	Homo sapiens	225-228	
2951244-1	1987	beta-Endorphin stimulates glycerol release from adipose tissue in vitro in the rabbit.	Glycerol	26-34	proopiomelanocortin	Homo sapiens	0-14	
6273120-7	1981	Moreover, the increased incorporation of labeled glycerol and palmitate into phospholipids suggests that ACTH and cAMP may stimulate the glycerol-3"-PO4 acyltransferase reaction.	Glycerol	49-57	proopiomelanocortin	Homo sapiens	105-109	
32549368-10	2020	Based on the test of ACTH release from semi-solid formulations and evaluation of rheological parameters, it was found that glycerol ointment is the most favourable base for ACTH.	Glycerol	123-131	proopiomelanocortin	Homo sapiens	173-177