PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26246380-5	2015	Furthermore, NaHS treatment led to suppression of fibroblast differentiation into myofibroblasts, and reduced the levels of collagen, TGF-beta1, and activated Smad3 in TGF-beta1-induced human cardiac fibroblasts in vitro.	sodium bisulfide	13-17	SMAD family member 3	Homo sapiens	159-164	
32171946-0	2020	Exogenous hydrogen sulfide donor NaHS alleviates nickel-induced epithelial-mesenchymal transition and the migration of A549 cells by regulating TGF-beta1/Smad2/Smad3 signaling.	sodium bisulfide	33-37	SMAD family member 3	Homo sapiens	160-165	
32171946-9	2020	Furthermore, exogenous NaHS alleviates the NiCl2-induced EMT and the migration ability of A549 cells only by regulating TGF-beta1/Smad2/Smad3, rather than the MAPK, signaling pathway.	sodium bisulfide	23-27	SMAD family member 3	Homo sapiens	136-141