PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25069981-10	2014	In addition, treatment of B. pertussis-infected mice with the carbonic anhydrase inhibitor acetazolamide reduced lung inflammatory pathology without affecting pendrin synthesis or bacterial loads.	Acetazolamide	91-104	solute carrier family 26, member 4	Mus musculus	159-166	
18971389-9	2008	(NH(4))(2)SO(4) and acetazolamide treatments reduced the relative number of pendrin-expressing cells in the collecting duct.	Acetazolamide	20-33	solute carrier family 26, member 4	Mus musculus	76-83	
18971389-13	2008	However, metabolic acidosis caused by acetazolamide or (NH(4))(2)SO(4) loading prevented the increase or even reduced pendrin expression despite low urinary Cl(-) excretion, suggesting an independent regulation by acid-base status.	Acetazolamide	38-51	solute carrier family 26, member 4	Mus musculus	118-125	
22116370-0	2011	Impact of bicarbonate, ammonium chloride, and acetazolamide on hepatic and renal SLC26A4 expression.	Acetazolamide	46-59	solute carrier family 26, member 4	Mus musculus	81-88	
22116370-11	2011	Slc26a4 transcript levels were significantly increased by NH(4)Cl and acetazolamide in liver, and significantly decreased by NH(4)Cl and by acetazolamide in kidney.	Acetazolamide	70-83	solute carrier family 26, member 4	Mus musculus	0-7	
22116370-11	2011	Slc26a4 transcript levels were significantly increased by NH(4)Cl and acetazolamide in liver, and significantly decreased by NH(4)Cl and by acetazolamide in kidney.	Acetazolamide	140-153	solute carrier family 26, member 4	Mus musculus	0-7	
22116370-12	2011	NH(4)Cl and acetazolamide reduced pendrin protein expression significantly in kidney, but did not significantly modify pendrin protein expression in liver.	Acetazolamide	12-25	solute carrier family 26, member 4	Mus musculus	34-41