PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29296191-0	2017	3-Methyladenine ameliorates liver fibrosis through autophagy regulated by the NF-kappaB signaling pathways on hepatic stellate cell.	3-methyladenine	0-15	nuclear factor kappa B subunit 1	Homo sapiens	78-87	
29296191-6	2017	Our studies proposed that 3-MA attenuates liver fibrosis induced by carbon tetrachloride (CCl4), and inhibit the expression of autophagy markers and transcriptional regulator NF-kappaB of hepatic stellate cell in vivo.	3-methyladenine	26-30	nuclear factor kappa B subunit 1	Homo sapiens	175-184	
29296191-8	2017	Conclusively, 3-MA ameliorates liver fibrosis through inhibition of autophagy regulated by the NF-kappaB signaling pathways on hepatic stellate cell.	3-methyladenine	14-18	nuclear factor kappa B subunit 1	Homo sapiens	95-104	
21262221-6	2011	Autophagic specific inhibitor 3-methyladenine (3-MA) treatment reversed silibinin-induced p53 suppression as well as NF-kappaB activation, suggesting that there was a positive feedback loop between p53 inhibition-mediated NF-kappaB activation and autophagy.	3-methyladenine	30-45	nuclear factor kappa B subunit 1	Homo sapiens	222-231	
21262221-6	2011	Autophagic specific inhibitor 3-methyladenine (3-MA) treatment reversed silibinin-induced p53 suppression as well as NF-kappaB activation, suggesting that there was a positive feedback loop between p53 inhibition-mediated NF-kappaB activation and autophagy.	3-methyladenine	47-51	nuclear factor kappa B subunit 1	Homo sapiens	117-126	
21262221-6	2011	Autophagic specific inhibitor 3-methyladenine (3-MA) treatment reversed silibinin-induced p53 suppression as well as NF-kappaB activation, suggesting that there was a positive feedback loop between p53 inhibition-mediated NF-kappaB activation and autophagy.	3-methyladenine	47-51	nuclear factor kappa B subunit 1	Homo sapiens	222-231	
34565256-5	2021	The results showed that LPS induced NF-kappaB-related pulmonary inflammation in observers and silicosis patients, as confirmed by an increase in the expression of IL-1beta, IL-6, TNF-alpha, and p65, which could be inhibited by 3-MA treatment.	3-methyladenine	227-231	nuclear factor kappa B subunit 1	Homo sapiens	36-45	
32555189-8	2020	Finally, 3-MA effectively blocked STAT3 and NF-kappaB phosphorylation and suppressed infiltration of macrophages and lymphocytes as well as release of multiple profibrogenic cytokines/chemokines in the injured kidney.	3-methyladenine	9-13	nuclear factor kappa B subunit 1	Homo sapiens	44-53	
28759800-7	2017	Moreover, autophagy inhibitor chloroquine (CQ) or 3-Methyladenine (3MA) treatment significantly suppressed phosphorylation and consequently degradation of NF-kappaB inhibitor I-kappaBalpha, reduced MM-MSCs-mediate activation of NF-kappaB and prevented MM-MSCs-induced resistance.	3-methyladenine	50-65	nuclear factor kappa B subunit 1	Homo sapiens	155-164	
28759800-7	2017	Moreover, autophagy inhibitor chloroquine (CQ) or 3-Methyladenine (3MA) treatment significantly suppressed phosphorylation and consequently degradation of NF-kappaB inhibitor I-kappaBalpha, reduced MM-MSCs-mediate activation of NF-kappaB and prevented MM-MSCs-induced resistance.	3-methyladenine	50-65	nuclear factor kappa B subunit 1	Homo sapiens	228-237	
28759800-7	2017	Moreover, autophagy inhibitor chloroquine (CQ) or 3-Methyladenine (3MA) treatment significantly suppressed phosphorylation and consequently degradation of NF-kappaB inhibitor I-kappaBalpha, reduced MM-MSCs-mediate activation of NF-kappaB and prevented MM-MSCs-induced resistance.	3-methyladenine	67-70	nuclear factor kappa B subunit 1	Homo sapiens	155-164	
28759800-7	2017	Moreover, autophagy inhibitor chloroquine (CQ) or 3-Methyladenine (3MA) treatment significantly suppressed phosphorylation and consequently degradation of NF-kappaB inhibitor I-kappaBalpha, reduced MM-MSCs-mediate activation of NF-kappaB and prevented MM-MSCs-induced resistance.	3-methyladenine	67-70	nuclear factor kappa B subunit 1	Homo sapiens	228-237	
24066693-7	2013	Moreover, TNF-alpha-induced NF-kappaB transactivation was suppressed by autophagy inhibitor 3-MA.	3-methyladenine	92-96	nuclear factor kappa B subunit 1	Homo sapiens	28-37	
22155150-7	2012	Autophagic inhibitor 3-methyladenine (3-MA) decreased phagocytosis and the expression of ERK whereas increased the expression of NF-kappaB- and caspase-1-mediated IL-1beta release.	3-methyladenine	21-36	nuclear factor kappa B subunit 1	Homo sapiens	129-138	
22155150-7	2012	Autophagic inhibitor 3-methyladenine (3-MA) decreased phagocytosis and the expression of ERK whereas increased the expression of NF-kappaB- and caspase-1-mediated IL-1beta release.	3-methyladenine	38-42	nuclear factor kappa B subunit 1	Homo sapiens	129-138	
21262221-6	2011	Autophagic specific inhibitor 3-methyladenine (3-MA) treatment reversed silibinin-induced p53 suppression as well as NF-kappaB activation, suggesting that there was a positive feedback loop between p53 inhibition-mediated NF-kappaB activation and autophagy.	3-methyladenine	30-45	nuclear factor kappa B subunit 1	Homo sapiens	117-126	
16644476-7	2006	Moreover, another PI3K inhibitor 3-methyladenine also strongly enhanced IL-1-induced NF-kappaB DNA binding, while LY303511, an inactive analogue of LY29, did not increase the NF-kappaB DNA binding.	3-methyladenine	33-48	nuclear factor kappa B subunit 1	Homo sapiens	85-94