PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21180121-0	2007	[Effects of omapatrilat on endothelin-1-induced proliferation and synthesis of cardiac fibroblasts].	omapatrilat	12-23	endothelin 1	Rattus norvegicus	27-39	
21180121-1	2007	AIM: To investigate the effects of omapatrilat (OMA) on endothelin-1-induced proliferation of cardiac fibroblasts (CFs).	omapatrilat	35-46	endothelin 1	Rattus norvegicus	56-68	
21180121-1	2007	AIM: To investigate the effects of omapatrilat (OMA) on endothelin-1-induced proliferation of cardiac fibroblasts (CFs).	omapatrilat	48-51	endothelin 1	Rattus norvegicus	56-68	
11390717-2	2001	The present study investigated the effect of the vasopeptidase inhibitor omapatrilat on endothelin-1 (ET-1)-mediated vascular function in salt-induced hypertension.	omapatrilat	73-84	endothelin 1	Rattus norvegicus	88-100	
11390717-2	2001	The present study investigated the effect of the vasopeptidase inhibitor omapatrilat on endothelin-1 (ET-1)-mediated vascular function in salt-induced hypertension.	omapatrilat	73-84	endothelin 1	Rattus norvegicus	102-106	
11390717-9	2001	While omapatrilat in vitro blunted the response to big endothelin-1 (big ET-1) and diminished ECE activity further (P<0.01 vs native segments), chronic treatment with omapatrilat in vivo restored contractions to ET-1 (120+/-6%) and big ET-1 (98+/-9%) in renal arteries, and therefore normalized renovascular ECE activity.	omapatrilat	6-17	endothelin 1	Rattus norvegicus	55-67	
11390717-9	2001	While omapatrilat in vitro blunted the response to big endothelin-1 (big ET-1) and diminished ECE activity further (P<0.01 vs native segments), chronic treatment with omapatrilat in vivo restored contractions to ET-1 (120+/-6%) and big ET-1 (98+/-9%) in renal arteries, and therefore normalized renovascular ECE activity.	omapatrilat	6-17	endothelin 1	Rattus norvegicus	73-77	
11390717-9	2001	While omapatrilat in vitro blunted the response to big endothelin-1 (big ET-1) and diminished ECE activity further (P<0.01 vs native segments), chronic treatment with omapatrilat in vivo restored contractions to ET-1 (120+/-6%) and big ET-1 (98+/-9%) in renal arteries, and therefore normalized renovascular ECE activity.	omapatrilat	170-181	endothelin 1	Rattus norvegicus	215-219	
11390717-9	2001	While omapatrilat in vitro blunted the response to big endothelin-1 (big ET-1) and diminished ECE activity further (P<0.01 vs native segments), chronic treatment with omapatrilat in vivo restored contractions to ET-1 (120+/-6%) and big ET-1 (98+/-9%) in renal arteries, and therefore normalized renovascular ECE activity.	omapatrilat	170-181	endothelin 1	Rattus norvegicus	215-219