PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21372496-3	2011	Treatment of tumor necrosis factor-alpha (TNF-alpha) suppressed the cell viability and facilitated the expression of Inter-Cellular Adhesion Molecule 1(ICAM-1) in human glomerular endothelial cells (HGECs) though, pretreatment of NO-NIF significantly recovered the TNF-alpha-induced cell damage to the same extent as Trolox-C did, and suppressed the ICAM-1 expression in a concentration dependent manner.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	317-325	tumor necrosis factor	Homo sapiens	13-40	
25148872-4	2014	Our results show that the pretreatment with well known antioxidants such as trolox and N-acetyl cysteine (NAC) partially reduced the apoptotic effects due to the administration of TNFalpha plus cycloheximide.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	76-82	tumor necrosis factor	Homo sapiens	180-188	
24488173-4	2014	The present results also show that the pretreatment with well-known antioxidants such as trolox and N-acetyl cysteine partially reduced the caspase activation caused by the administration of TNFalpha.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	89-95	tumor necrosis factor	Homo sapiens	191-199	
21372496-3	2011	Treatment of tumor necrosis factor-alpha (TNF-alpha) suppressed the cell viability and facilitated the expression of Inter-Cellular Adhesion Molecule 1(ICAM-1) in human glomerular endothelial cells (HGECs) though, pretreatment of NO-NIF significantly recovered the TNF-alpha-induced cell damage to the same extent as Trolox-C did, and suppressed the ICAM-1 expression in a concentration dependent manner.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	317-325	tumor necrosis factor	Homo sapiens	42-51	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	266-321	tumor necrosis factor	Homo sapiens	26-53	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	266-321	tumor necrosis factor	Homo sapiens	55-64	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	266-321	tumor necrosis factor	Homo sapiens	55-58	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	323-329	tumor necrosis factor	Homo sapiens	26-53	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	323-329	tumor necrosis factor	Homo sapiens	55-64	
19289607-7	2009	Treatment of neurons with tumor necrosis factor-alpha (TNF-alpha) stimulated reactive oxygen species generation and resulted in the accumulation of tau-Citrine and tau-Cerulean in neurites, which was inhibited by neutralization of TNF and the free radical inhibitor 6-hydroxy-2,5,7,8-tetramethylchromane-2-carboxylic acid (Trolox).	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	323-329	tumor necrosis factor	Homo sapiens	55-58	
16910777-7	2006	The lipid hydroperoxide scavengers, beta-hydroxytoluene and trolox, and the iron chelator, desferroxamine, showed partial recovery of TNF-induced apoptosis.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	60-66	tumor necrosis factor	Homo sapiens	134-137	
14555462-11	2004	In human monocytes, verapamil, Trolox, BAPTA-AM, and W-7 reduced Uf-stimulated TNF-alpha protein release.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	31-37	tumor necrosis factor	Homo sapiens	79-88	
11298495-5	2001	Scavenging of ROS by means of the antioxidant trolox prevented the TMT-induced release of TNF-alpha from glial cells, while indomethacin was found to suppress ROS formation induced by 1 microM TMT in glial cells.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	46-52	tumor necrosis factor	Homo sapiens	90-99