PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17341418-12	2007	Furthermore, Trolox attenuated indomethacin-induced increased phosphorylation in ERK, p38 MAPK, JNK, and AKT.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	13-19	mitogen-activated protein kinase 8	Homo sapiens	96-99	
24938881-9	2014	Abrin also shown to increase in stress factor associated proteins SAPK/JNK, c-fos and c-jun levels which were effectively suppressed by NAC and trolox.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	144-150	mitogen-activated protein kinase 8	Homo sapiens	71-74	
20381862-7	2010	Compared to SPIO, USPIO induced more pronounced biochemical alterations and cytotoxicity, which could be antagonized by the JNK inhibitor V. Alternatively, treatment of macrophages with Trolox or N-acetyl-L-cysteine, two functionally different scavengers of reactive oxygen species, abolished both the JNK activation and the subsequent cytotoxic effects.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	186-192	mitogen-activated protein kinase 8	Homo sapiens	124-127	
15466933-4	2005	Treatment with As(2)O(3) and trolox increased intracellular oxidative stress, as evidenced by heme oxygenase-1 (HO-1) protein levels, c-Jun terminal kinase (JNK) activation, and protein and lipid oxidation.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	29-35	mitogen-activated protein kinase 8	Homo sapiens	134-155	
15466933-4	2005	Treatment with As(2)O(3) and trolox increased intracellular oxidative stress, as evidenced by heme oxygenase-1 (HO-1) protein levels, c-Jun terminal kinase (JNK) activation, and protein and lipid oxidation.	6-hydroxy-2,5,7,8-tetramethylchroman-2-carboxylic acid	29-35	mitogen-activated protein kinase 8	Homo sapiens	157-160