PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2891847-4	1988	Diltiazem also inhibited mesenteric vasoconstrictor responses to angiotensin II, vasopressin, prostaglandin F2 alpha and KCl.	Diltiazem	0-9	arginine vasopressin	Homo sapiens	81-92	
1782723-0	1991	Calcium-channel blocking agents verapamil and diltiazem are inhibitors of vasopressin-induced human platelet activation.	Diltiazem	46-55	arginine vasopressin	Homo sapiens	74-85	
1782723-2	1991	This study investigated the influences of calcium-channel blocking agents verapamil and diltiazem on platelet responses induced by arginine vasopressin (AVP) and lysine vasopressin (LVP).	Diltiazem	88-97	arginine vasopressin	Homo sapiens	140-151	
1782723-2	1991	This study investigated the influences of calcium-channel blocking agents verapamil and diltiazem on platelet responses induced by arginine vasopressin (AVP) and lysine vasopressin (LVP).	Diltiazem	88-97	arginine vasopressin	Homo sapiens	169-180	
1782723-10	1991	Our data provide evidence that the effects of verapamil and diltiazem on vasopressin-induced platelet responses may be directly related to inhibition of extracellular calcium entry.	Diltiazem	60-69	arginine vasopressin	Homo sapiens	73-84	
2279694-4	1990	Results showed that diltiazem inhibits platelet aggregation induced by ADP, arginine vasopressin, adrenaline, collagen, Na arachidonate, thrombin and phorbol ester PMA in a dose-dependent way.	Diltiazem	20-29	arginine vasopressin	Homo sapiens	85-96	
4044600-9	1985	This reactivation of phosphorylase showed saturation kinetics with respect to extracellular [Ca2+], was maximally stimulated within 1 min of vasopressin addition and was inhibited by high concentration of diltiazem.	Diltiazem	205-214	arginine vasopressin	Homo sapiens	141-152	
6299112-3	1983	D600 (200 microM), diltiazem (200 microM), and nifedipine (60 microM) inhibited vasopressin-induced water flow but enhanced adenosine 3",5"-cyclic monophosphate (cAMP)-induced water flow, suggesting that the drugs inhibit cAMP generation in response to vasopressin but enhance the response to exogenous cAMP by inhibiting phosphodiesterase activity.	Diltiazem	19-28	arginine vasopressin	Homo sapiens	80-91