PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 32399331-6 2020 We postulate that elevated CRP may warrant steroid therapy over conservative management and could lead to a possible decreased hospital stay. Steroids 43-50 C-reactive protein Homo sapiens 27-30 31858957-10 2020 Higher CRP values at 6 months appear to be associated with a higher risk of longer steroid therapy. Steroids 83-90 C-reactive protein Homo sapiens 7-10 31806506-7 2020 RESULTS: The steroid group showed significantly lower CRP levels on POD 2 (P < .05) and POD 4 (P < .05) but a higher CRP level on POD 6 (P < .05). Steroids 13-20 C-reactive protein Homo sapiens 54-57 31806506-7 2020 RESULTS: The steroid group showed significantly lower CRP levels on POD 2 (P < .05) and POD 4 (P < .05) but a higher CRP level on POD 6 (P < .05). Steroids 13-20 C-reactive protein Homo sapiens 123-126 31806506-9 2020 Peak values in CRP and ESR in the steroid group (POD 4 and 6) appeared two days later compared with the non-steroid group (POD 2 and 4). Steroids 34-41 C-reactive protein Homo sapiens 15-18 33086971-5 2020 Initially, steroid administration achieved remission, but her symptoms and high CRP levels relapsed every time the steroid dosage was tapered down. Steroids 115-122 C-reactive protein Homo sapiens 80-83 33242860-7 2020 After the steroid pulse therapy, the C-reactive protein (CRP) decreased from 6.47 mg/dL to 0.76 mg/dL, but there was little improvement in the symptoms, and the CRP then increased to 4.44 mg/dL. Steroids 10-17 C-reactive protein Homo sapiens 37-55 31064976-6 2019 CASE REPORT We describe a case of 38-year-old female with diffuse musculoskeletal pain and elevated ESR and CRP who was initially misdiagnosed with PMR and responded partially to steroids. Steroids 179-187 C-reactive protein Homo sapiens 108-111 31065018-5 2019 Compared with placebo groups, steroids application could effectively relieve pain on postoperative day (POD)1; decrease C-Reactive protein (CRP) level on POD3; improve range of motion (ROM) in postoperative 5 days; reduce morphine consumption, achieve earlier straight leg raising (SLR), and shorten the length of stay (LOS) in hospital. Steroids 30-38 C-reactive protein Homo sapiens 120-138 31065018-5 2019 Compared with placebo groups, steroids application could effectively relieve pain on postoperative day (POD)1; decrease C-Reactive protein (CRP) level on POD3; improve range of motion (ROM) in postoperative 5 days; reduce morphine consumption, achieve earlier straight leg raising (SLR), and shorten the length of stay (LOS) in hospital. Steroids 30-38 C-reactive protein Homo sapiens 140-143 30268443-8 2019 RESULTS: Compared to the non-steroid group, patients receiving dexamethasone yielded a significantly decreased CRP on POD0 (5.5 vs 8.3), POD1 (11.8 vs 22.0), and POD2 (85.5 vs 148.9). Steroids 29-36 C-reactive protein Homo sapiens 111-114 30804146-0 2019 Postoperative Level of C-Reactive Protein Is a Prognosticator After Esophageal Cancer Surgery With Perioperative Steroid Therapy and Enhanced Recovery After Surgery Care. Steroids 113-120 C-reactive protein Homo sapiens 23-41 30804146-7 2019 CONCLUSION: The present results suggest that the postoperative CRP level can be a prognosticator in patients with esophageal cancer who have received perioperative steroid therapy and ERAS care. Steroids 164-171 C-reactive protein Homo sapiens 63-66 26865349-7 2016 Similarly, significant reductions were noted in steroid doses within groups at 3 months (P < 0.05) and 12 months (P < 0.05), with notable reductions in C-reactive protein (CRP) at 3 months within groups (P < 0.05). Steroids 48-55 C-reactive protein Homo sapiens 178-181 28737646-9 2018 Receiver operating characteristic statistics were used to determine the optimal stool frequency, CRP, albumin, and CRP/albumin ratio (CAR) to predict steroid response. Steroids 150-157 C-reactive protein Homo sapiens 115-118 27938961-11 2016 The initiation of steroids decreased the diagnostic utility of the ESR, CRP, and platelets values (AUC = 0.58, 0.61, and 0.63, respectively). Steroids 18-26 C-reactive protein Homo sapiens 72-75 26925610-10 2016 After adjustment for the cumulative duration of steroid therapy, these 2 correlations remained significant (CRP: P = 0.0008; orosomucoid: P < 0.0001). Steroids 48-55 C-reactive protein Homo sapiens 108-111 27726926-10 2016 Steroid treatment should be reserved for patients who present with major pain, nerve lesions, associated autoimmune disease, or elevated C reactive protein or CK. Steroids 0-7 C-reactive protein Homo sapiens 137-155 30498759-7 2018 Higher CRP, ESR level, RF positivity, and the presence of fever or vasculitic skin lesions at the time of diagnosis indicated a higher steroid demand and the administration of higher number of immunosuppressants during the follow-up within anti-Jo-1 positive patients. Steroids 135-142 C-reactive protein Homo sapiens 7-10 30178473-7 2018 After accounting for the effects of steroids, CRP was higher in carbamazepine patients than in other patients. Steroids 36-44 C-reactive protein Homo sapiens 46-49 29074374-0 2018 C-Reactive Protein Levels at Diagnosis of Acute Graft-versus-Host Disease Predict Steroid-Refractory Disease, Treatment-Related Mortality, and Overall Survival after Allogeneic Hematopoietic Stem Cell Transplantation. Steroids 82-89 C-reactive protein Homo sapiens 0-18 29074374-4 2018 Because C-reactive protein (CRP) is a systemic inflammatory marker, we aimed to investigate whether plasma CRP concentrations at the diagnosis of aGVHD can predict the risk of failing first-line therapy and developing steroid-refractory disease. Steroids 218-225 C-reactive protein Homo sapiens 107-110 29074374-10 2018 CRP levels were significantly higher in patients who developed steroid-refractory disease compared with those who responded to high-dose corticosteroid therapy (odds ratio, 1.50; 95% confidence interval, 1.18-1.93; P = .001). Steroids 63-70 C-reactive protein Homo sapiens 0-3 29074374-13 2018 These results suggest that CRP level at diagnosis is a valid predictor of the development of steroid-refractory disease in patients who develop grade II-IV aGVHD after HSCT. Steroids 93-100 C-reactive protein Homo sapiens 27-30 29340241-7 2018 Binary logistic regression analysis for factors including age, BMI, creatinine, hs-CRP, smoking, steroid use, and forced expiratory volume in one second (FEV1) revealed that an hs-CRP level >=5 and decreased creatinine level were independent risk factors for osteoporosis in COPD patients. Steroids 97-104 C-reactive protein Homo sapiens 180-183 29340241-8 2018 Lower BMI tended to be associated with osteoporosis development, although it did not reach statistical significance, and hs-CRP was associated with COPD severity and steroid use history. Steroids 166-173 C-reactive protein Homo sapiens 124-127 29202716-0 2017 Prediction of postoperative inflammatory complications after esophageal cancer surgery based on early changes in the C-reactive protein level in patients who received perioperative steroid therapy and enhanced recovery after surgery care: a retrospective analysis. Steroids 181-188 C-reactive protein Homo sapiens 117-135 29202716-11 2017 CONCLUSIONS: CRP value on POD 4 may be useful for predicting SICs in esophageal cancer patients who receive radical esophagectomy with perioperative steroid therapy and ERAS care. Steroids 149-156 C-reactive protein Homo sapiens 13-16 29403332-3 2017 The second describes the use of c-reactive protein as a marker for starting treatment with steroids in children with orbital cellulitis. Steroids 91-99 C-reactive protein Homo sapiens 32-50 28237892-0 2017 Increased C-reactive protein levels at diagnosis negatively predict platelet count recovery after steroid-treatment in newly diagnosed adult immune thrombocytopenia patients. Steroids 98-105 C-reactive protein Homo sapiens 10-28 27021506-7 2016 RESULTS: A high CRP was associated with younger age, steroid use, colonic or ileocolonic location, high CD activity index, and active inflammation at colonoscopy (p<0.001). Steroids 53-60 C-reactive protein Homo sapiens 16-19 24847348-10 2014 Though the true relationship between MG and TA was not revealed in present case, considering the fact that complete remission of nephrotic syndrome occurred following the improvement of C-reactive protein level in response to steroid therapy, TA might be the secondary cause of MG. To our best knowledge, only two case reports described the association of MG and TA previously. Steroids 226-233 C-reactive protein Homo sapiens 186-204 25683977-8 2015 Maximum body temperature and C-reactive protein were significantly decreased in the moderate-dose steroid group compared to the other groups (p<0.01). Steroids 98-105 C-reactive protein Homo sapiens 29-47 24655315-8 2015 Serum total bilirubin, interleukin-6 and C-reactive protein were significantly lower in the steroid than in the control group on postoperative day 1 (P = 0.02, 0.004 and 0.02, respectively). Steroids 92-99 C-reactive protein Homo sapiens 41-59 25393908-0 2015 C-Reactive Protein As a Marker for Initiating Steroid Treatment in Children With Orbital Cellulitis. Steroids 46-53 C-reactive protein Homo sapiens 0-18 25393908-12 2015 The average CRP at the onset of steroid treatment was 2.8 mg/dl (range: 0.5-4). Steroids 32-39 C-reactive protein Homo sapiens 12-15 25393908-23 2015 Patients who received systemic steroids after CRP dropped below 4 mg/dl were discharged from the hospital earlier than patients who did not receive systemic steroids. Steroids 31-39 C-reactive protein Homo sapiens 46-49 24824230-0 2014 Increased C-reactive protein is associated with airway wall thickness in steroid-naive asthma. Steroids 73-80 C-reactive protein Homo sapiens 10-28 24495103-12 2014 CONCLUSIONS: FC, CRP, and SCCAI seem to be reliable markers of treatment response during steroid treatment. Steroids 89-96 C-reactive protein Homo sapiens 17-20 24390062-7 2014 Clinical remission at week 12 was achieved in 155 (73%) patients; both exclusive enteral nutrition and steroids were associated with normal CRP remission at week 12, although in a post hoc subgroup analysis exclusive enteral nutrition was superior in mild-to-moderate disease for this outcome. Steroids 103-111 C-reactive protein Homo sapiens 140-143 24390062-8 2014 Among those in steroid-free remission in week 12, normal CRP predicted 1-year sustained remission (86% for normal CRP versus 61% for elevated CRP; P = 0.02). Steroids 15-22 C-reactive protein Homo sapiens 57-60 24390062-10 2014 CONCLUSIONS: Normal CRP steroid-free remission at week 12 was impacted by type of induction therapy, but not by early immunomodulation. Steroids 24-31 C-reactive protein Homo sapiens 20-23 23339056-9 2013 In addition, steroids significantly reduced postoperative blood levels of bilirubin, and of inflammatory markers such as IL-6 and C-reactive protein. Steroids 13-21 C-reactive protein Homo sapiens 130-148 23644895-12 2013 The CRP levels in steroids groups were also lower than in control group on the third day. Steroids 18-26 C-reactive protein Homo sapiens 4-7 23381198-4 2013 We hypothesized that preoperative steroid administration, routinely used only in conventional CABG, may alleviate high postoperative PAF and CRP levels. Steroids 34-41 C-reactive protein Homo sapiens 141-144 23381198-11 2013 CONCLUSIONS: Preoperative steroid administration in OPCABG patients significantly suppresses CRP elevation and prevents postoperative PAF without increasing in-hospital mortality or infectious complications. Steroids 26-33 C-reactive protein Homo sapiens 93-96 23672831-7 2013 The area under the receiver operating characteristic curve values for association with steroid-free sustained remission were 0.70 for the PUCAI (95% confidence interval [CI], 0.53-0.88), 0.56 for mucosal healing (95% CI, 0.36-0.76), and 0.44 for level of CRP (95% CI, 0.24-0.65). Steroids 87-94 C-reactive protein Homo sapiens 255-258 22829554-5 2013 Meta-analysis showed that: 1) steroid group had lower serum IL-6 level on POD 1 and POD 3 (p<0.05), and 2) steroid group had lower serum CRP level on POD 3 (p<0.05) with no significant difference on POD 1 and POD 7. Steroids 30-37 C-reactive protein Homo sapiens 140-143 23448790-13 2013 C-reactive protein drop to normal after infliximab induction was predictive of steroid-free remission at 6 (HR, 5.9) and 12 months (HR, 4.6) and steroid-free remission and MH at 12 months (odds ratio, 6.0). Steroids 79-86 C-reactive protein Homo sapiens 0-18 23448790-13 2013 C-reactive protein drop to normal after infliximab induction was predictive of steroid-free remission at 6 (HR, 5.9) and 12 months (HR, 4.6) and steroid-free remission and MH at 12 months (odds ratio, 6.0). Steroids 145-152 C-reactive protein Homo sapiens 0-18 22829554-5 2013 Meta-analysis showed that: 1) steroid group had lower serum IL-6 level on POD 1 and POD 3 (p<0.05), and 2) steroid group had lower serum CRP level on POD 3 (p<0.05) with no significant difference on POD 1 and POD 7. Steroids 110-117 C-reactive protein Homo sapiens 140-143 22745302-11 2012 The C-reactive protein level on POD 2 was lower in the steroid group (P < 0.005). Steroids 55-62 C-reactive protein Homo sapiens 4-22 23155348-4 2012 The patient received antibiotics and other supportive therapy, and the postoperative course was uneventful, however, elevated levels of serum IgG, IgG4 and C-reactive protein were noted, which normalized after the introduction of steroid therapy. Steroids 230-237 C-reactive protein Homo sapiens 156-174 21457289-8 2012 The patient"s QTc, amylase and C-reactive protein also fell following initiation of steroid treatment. Steroids 84-91 C-reactive protein Homo sapiens 31-49 22628929-17 2012 CONCLUSIONS: It was found that serum concentrations of hs-CRP significantly increased in asthmatic patients, and in the steroid-native group it partly correlated with FEV(1) and numbers of sputum eosinophils. Steroids 120-127 C-reactive protein Homo sapiens 58-61 19836939-9 2009 RESULTS: There was an increase of high sensitivity C-reactive protein in asthmatic patients among both steroid inhaled and steroid naive patients compared to the healthy controls. Steroids 103-110 C-reactive protein Homo sapiens 51-69 21423006-4 2011 Simple clinical and laboratory measures predict steroid refractoriness (such as stool frequency 3-8/day and C-reactive protein > 45 mg/l on day 3) and salvage therapy is appropriate at this stage. Steroids 48-55 C-reactive protein Homo sapiens 108-126 21933279-9 2012 All patients with PES had high serum levels of C-reactive protein (CRP), which were significantly reduced after effective steroid treatment. Steroids 122-129 C-reactive protein Homo sapiens 47-65 21933279-9 2012 All patients with PES had high serum levels of C-reactive protein (CRP), which were significantly reduced after effective steroid treatment. Steroids 122-129 C-reactive protein Homo sapiens 67-70 21736395-7 2011 There was a statistically significant difference(p < 0.01) in CRP serum levels from 21 to 8 mg/dl at week 48.Nine out of fifteen patients (60%) treated with corticosteroids were able to discontinue steroids. Steroids 167-175 C-reactive protein Homo sapiens 65-68 21223500-0 2011 Prognostic usefulness of high sensitivity C-reactive protein for transforaminal epidural steroid injection in patients with radicular pain. Steroids 89-96 C-reactive protein Homo sapiens 42-60 21223500-6 2011 The purpose of this study was to evaluate the usefulness of high sensitivity C-reactive protein (hsCRP) as a marker for predicting the efficacy of lumbar transforaminal epidural steroid injection. Steroids 178-185 C-reactive protein Homo sapiens 77-95 19836939-9 2009 RESULTS: There was an increase of high sensitivity C-reactive protein in asthmatic patients among both steroid inhaled and steroid naive patients compared to the healthy controls. Steroids 123-130 C-reactive protein Homo sapiens 51-69 19836939-10 2009 Serum high sensitivity C-reactive protein correlated negatively with pulmonary function tests and positively with sputum eosinophil % in both inhaled steroid and steroid naive groups. Steroids 150-157 C-reactive protein Homo sapiens 23-41 19836939-10 2009 Serum high sensitivity C-reactive protein correlated negatively with pulmonary function tests and positively with sputum eosinophil % in both inhaled steroid and steroid naive groups. Steroids 162-169 C-reactive protein Homo sapiens 23-41 18508833-9 2008 In conclusion, consecutive C-reactive protein measurements are useful in the first week in follow-up of antibiotic treatment for severe community-acquired pneumonia when taking the causative microorganism and use of steroids into account. Steroids 216-224 C-reactive protein Homo sapiens 27-45 19398195-8 2009 RESULTS: Independent clinical predictors for steroid response vs. non-steroid response in biopsy proven AR were "days after transplantation" (p=0.01, adjusted hazard ratio (HR) 1.2), "decline in home spirometry slope" (p=0.03, HR 0.97), "adherence to home spirometry" (p=0.05, HR 0.98) and "serum CRP" (p=0.02, HR 0.87). Steroids 45-52 C-reactive protein Homo sapiens 297-301 19851514-9 2009 A small reduction of CRP levels after NSAIDs use and promptly ameliorated CRP and symptoms by a low-dose steroid therapy, which was commonly observed in patients with PMR, were also found in a patient with RS3PE syndrome. Steroids 105-112 C-reactive protein Homo sapiens 21-24 19851514-9 2009 A small reduction of CRP levels after NSAIDs use and promptly ameliorated CRP and symptoms by a low-dose steroid therapy, which was commonly observed in patients with PMR, were also found in a patient with RS3PE syndrome. Steroids 105-112 C-reactive protein Homo sapiens 74-77 18453363-3 2008 Decreases in inflammatory cytokines, C-reactive protein, and inflammatory cells have been observed with corticosteroid use, suggesting a possible mechanism for a therapeutic benefit of steroids. Steroids 185-193 C-reactive protein Homo sapiens 37-55 18341528-14 2008 CONCLUSIONS: Our findings show that in postmenopausal women high levels of endogenous oestrogenic and androgenic sex steroids coincide with high CRP levels. Steroids 117-125 C-reactive protein Homo sapiens 145-148 18330505-0 2008 Influence of steroids on procalcitonin and C-reactive protein in patients with COPD and community-acquired pneumonia. Steroids 13-21 C-reactive protein Homo sapiens 43-61 17130328-7 2006 The peak white cell counts and C-reactive protein levels after the 7(th) postoperative day were significantly higher in the steroid group. Steroids 124-131 C-reactive protein Homo sapiens 31-49 16678922-7 2007 RESULTS: In both genders, we observed inverse associations between estimated VO2max and levels of CRP after controlling for age, race, body mass index category, hypertension, diabetes, smoking status, alcohol consumption, and use of medications including aspirin, non-steroidal anti-inflammatory drugs, steroid, lipid-lowering agents, antimicrobials, or estrogen/progesterone (in women). Steroids 268-275 C-reactive protein Homo sapiens 98-101 17222316-6 2007 Statistical analysis was performed using the Kaplan-Meier method and multivariable Cox proportional hazard regression model taking into consideration gender, age at diagnosis, disease location and behavior, smoking habits, CDAI score before steroid treatment, and C reactive protein values at steroid weaning, as covariates. Steroids 293-300 C-reactive protein Homo sapiens 264-282 17222316-11 2007 At multivariate analysis, increased C reactive protein at steroid weaning and penetrating complications were independent risk factors for further steroid requirement (OR 5.57, 95% CI 1.20-25.91, P= 0.001 and OR 4.20, 95% CI 1.76-10.04, P= 0.005, respectively). Steroids 58-65 C-reactive protein Homo sapiens 36-54 17222316-11 2007 At multivariate analysis, increased C reactive protein at steroid weaning and penetrating complications were independent risk factors for further steroid requirement (OR 5.57, 95% CI 1.20-25.91, P= 0.001 and OR 4.20, 95% CI 1.76-10.04, P= 0.005, respectively). Steroids 146-153 C-reactive protein Homo sapiens 36-54 17222316-12 2007 CONCLUSION: Despite an initial clinical response and successful steroid tapering, 35% of patients required further steroid treatment within 1 yr. An increased C reactive protein value, at steroid weaning, despite clinical remission, and penetrating complications may predict further steroid requirement in already steroid responsive patients. Steroids 64-71 C-reactive protein Homo sapiens 159-177 17222316-12 2007 CONCLUSION: Despite an initial clinical response and successful steroid tapering, 35% of patients required further steroid treatment within 1 yr. An increased C reactive protein value, at steroid weaning, despite clinical remission, and penetrating complications may predict further steroid requirement in already steroid responsive patients. Steroids 115-122 C-reactive protein Homo sapiens 159-177 17222316-12 2007 CONCLUSION: Despite an initial clinical response and successful steroid tapering, 35% of patients required further steroid treatment within 1 yr. An increased C reactive protein value, at steroid weaning, despite clinical remission, and penetrating complications may predict further steroid requirement in already steroid responsive patients. Steroids 115-122 C-reactive protein Homo sapiens 159-177 17222316-12 2007 CONCLUSION: Despite an initial clinical response and successful steroid tapering, 35% of patients required further steroid treatment within 1 yr. An increased C reactive protein value, at steroid weaning, despite clinical remission, and penetrating complications may predict further steroid requirement in already steroid responsive patients. Steroids 115-122 C-reactive protein Homo sapiens 159-177 17222316-12 2007 CONCLUSION: Despite an initial clinical response and successful steroid tapering, 35% of patients required further steroid treatment within 1 yr. An increased C reactive protein value, at steroid weaning, despite clinical remission, and penetrating complications may predict further steroid requirement in already steroid responsive patients. Steroids 115-122 C-reactive protein Homo sapiens 159-177 17308218-7 2007 ESR, CRP and haemoglobin improved in the steroid group only (p<0.05). Steroids 41-48 C-reactive protein Homo sapiens 5-8 17379997-11 2007 Steroid therapy including methylprednisolone pulse therapy (500 mg/day, 3 days) and 2 courses of plasma exchange were effective in reducing the fever, anti-GBM antibody titer and C-reactive protein level. Steroids 0-7 C-reactive protein Homo sapiens 179-197 17909718-8 2007 RESULTS: Postoperative plasma concentrations of IL-6 (days 1 and 2), IL-8 (days 2 and 3), and CRP (days 1-4) were significantly lower in the steroid than in the control group. Steroids 141-148 C-reactive protein Homo sapiens 94-97 17118943-4 2007 PE and steroid pulse therapy reduced his plasma CRP level. Steroids 7-14 C-reactive protein Homo sapiens 48-51 16405429-11 2006 CRP (6.1 mg/dL vs. 3.1 mg/dL; P < 0.001) and IL-6 (295 ng/mL vs. 122 ng/mL; P = 0.001) were decreased during steroid therapy, but not PCT (2.3 ng/dL vs. 2.0 ng/dL; n.s.). Steroids 112-119 C-reactive protein Homo sapiens 0-3 16707391-5 2006 Serum hs-CRP levels were significantly increased in steroid-naive patients (mean+/-sd 1.33+/-1.48 mg.L(-1)) compared with controls (0.21+/-0.30 mg.L(-1)), but not in patients on inhaled corticosteroid. Steroids 52-59 C-reactive protein Homo sapiens 9-12 16707391-6 2006 Among steroid-naive patients, serum hs-CRP levels significantly negatively correlated with indices of pulmonary function (forced expiratory volume in one second/forced vital capacity and forced mid-expiratory flow) and positively with sputum eosinophil count. Steroids 6-13 C-reactive protein Homo sapiens 39-42 14669391-1 2003 UNLABELLED: The main markers of intrauterine infection: C-reactive protein and white blood cell count are dependent on medical management as steroids or antibiotics therapy. Steroids 141-149 C-reactive protein Homo sapiens 56-74 16190914-7 2005 Peak serum IL-6 and CRP concentrations were significantly lower in the steroid treatment group than in the non-steroid treatment group. Steroids 71-78 C-reactive protein Homo sapiens 20-23 16285590-10 2005 C-reactive protein and MPO-ANCA decreased to normal levels after the initial steroid therapy, but serum amyloid A protein (SAA) gradually elevated. Steroids 77-84 C-reactive protein Homo sapiens 0-18 15750272-6 2005 After steroid therapy was restarted, there were improvements in her audibility, radial arterial pulsation, and levels of inflammatory markers (erythrocyte sedimentation rate, C-reactive protein, and gamma-globulin), fibrinogen, interleukin-6, and RANTES (regulated on activation, normal T cell expressed and secreted). Steroids 6-13 C-reactive protein Homo sapiens 175-193 14751958-0 2004 Raised concentrations of C reactive protein in anabolic steroid using bodybuilders. Steroids 56-63 C-reactive protein Homo sapiens 25-43 14751958-1 2004 OBJECTIVE: To examine levels of C reactive protein in users of anabolic androgenic steroids (AAS) compared with age matched control groups consisting of AAS using (but abstinent)/resistance trained and non-drug using/sedentary controls. Steroids 83-91 C-reactive protein Homo sapiens 32-50 15678964-11 2005 The serum CRP on the 3rd postoperative day was significantly lower in the steroid group than in control (8.5 mg/dl versus 13.3 mg/dl, p=0.011) while it was not different between those 2 groups on the 7th postoperative day. Steroids 74-81 C-reactive protein Homo sapiens 10-13 17670296-5 2004 We conclude that steroids may reduce Troponin I release, CRP and reduce Interleukin-6. Steroids 17-25 C-reactive protein Homo sapiens 57-60 15042565-6 2004 Serum creatinine level decreased, and C-reactive protein turned negative after steroid therapy. Steroids 79-86 C-reactive protein Homo sapiens 38-56 12799248-10 2003 Then the steroid therapy was gradually tapered, guided primarily by the levels of ESR and CRP. Steroids 9-16 C-reactive protein Homo sapiens 90-93 12955709-9 2003 Intravenous steroid pulse therapy followed by oral prednisolone (PSL) was effective for reducing the fever, eosinophilia, anti-GBM antibody titer, and C-reactive protein level, but did not improve renal function because renal tissue already was irreversibly damaged. Steroids 12-19 C-reactive protein Homo sapiens 151-169 12792130-9 2003 We found that the most reliable and sensitive parameters to regulate and taper down steroid therapy were the levels of erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP) and NOT systemic symptoms. Steroids 84-91 C-reactive protein Homo sapiens 160-178 12792130-9 2003 We found that the most reliable and sensitive parameters to regulate and taper down steroid therapy were the levels of erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP) and NOT systemic symptoms. Steroids 84-91 C-reactive protein Homo sapiens 180-183 8367755-5 1993 We suggest using the C-reactive protein level as a monitor of subclinical disease activity; it is therefore beneficial in the adjustment of steroid therapy. Steroids 140-147 C-reactive protein Homo sapiens 21-39 12147770-14 2002 Steroid-treated patients showed less favourable nutritional conditions, with lower serum albumin and body mass index vs non-steroid-treated patients; moreover, C-reactive protein values were persistently higher in the steroid-treated group. Steroids 0-7 C-reactive protein Homo sapiens 160-178 12147770-14 2002 Steroid-treated patients showed less favourable nutritional conditions, with lower serum albumin and body mass index vs non-steroid-treated patients; moreover, C-reactive protein values were persistently higher in the steroid-treated group. Steroids 218-225 C-reactive protein Homo sapiens 160-178 11377237-9 2001 However, about 2 weeks after the start of steroid administration, a fever and a further elevation of CRP were observed with an increase of beta-D-glucan in serum. Steroids 42-49 C-reactive protein Homo sapiens 101-104 10688417-6 1999 C-reactive protein values at one postoperative day in the steroid group were also significantly lower than those in the control group. Steroids 58-65 C-reactive protein Homo sapiens 0-18 9341289-6 1997 After treatment with on oral steroid drug the serum KL-6 level decreased gradually, symptoms were relieved the previously high level of c-reactive protein in serum decreased, the previously high white blood cell count decreased, and radiographic findings returned to normal. Steroids 29-36 C-reactive protein Homo sapiens 136-154 8803706-7 1996 The value of the C-reactive protein at postoperative day 3 in the steroid pulse group was significantly lower than that in the control group, and the postoperative peak value of bilirubin as well as the incidence of postoperative complications in the control group tended to be higher than those in the steroid pulse group. Steroids 66-73 C-reactive protein Homo sapiens 17-35 8803706-7 1996 The value of the C-reactive protein at postoperative day 3 in the steroid pulse group was significantly lower than that in the control group, and the postoperative peak value of bilirubin as well as the incidence of postoperative complications in the control group tended to be higher than those in the steroid pulse group. Steroids 303-310 C-reactive protein Homo sapiens 17-35 8196250-9 1994 The steroid therapy has been continued and adjusted by the values of CRP as well as the cutaneous and mucous manifestations. Steroids 4-11 C-reactive protein Homo sapiens 69-72 11231856-6 2001 RESULTS: Postoperative levels of serum and peritoneal IL-6 and levels of C-reactive protein were significantly lower in the steroid group than in controls. Steroids 124-131 C-reactive protein Homo sapiens 73-91 11231856-11 2001 CONCLUSIONS: Preoperative steroid administration significantly elevated anti-inflammatory cytokine IL-10 levels, suppressed the levels of inflammatory cytokines IL-6 and C-reactive protein, and prevented postoperative elevation of total bilirubin values. Steroids 26-33 C-reactive protein Homo sapiens 170-188 11030213-6 2000 Treatment with a steroid improved the deep sensory disturbance, unsteady gait, and CRP level. Steroids 17-24 C-reactive protein Homo sapiens 83-86 10459204-4 1999 NicoCard CRP kit manufactured by the Nicomed Firm allows a rapid (2 min) and accurate (quantitative using NicoCard Readers) detection of inflammation and evaluation of its severity, helps differentiate between bacterial and viral infections, choose adequate therapy (antibiotics, steroids, antiinflammatory agents), and monitor the treatment efficacy. Steroids 280-288 C-reactive protein Homo sapiens 9-12 9605127-1 1998 In vitro, IL-6 is the main inducer of the human C-reactive protein (CRP) gene, and IL-1 and steroids can enhance this effect. Steroids 92-100 C-reactive protein Homo sapiens 48-66 9605127-1 1998 In vitro, IL-6 is the main inducer of the human C-reactive protein (CRP) gene, and IL-1 and steroids can enhance this effect. Steroids 92-100 C-reactive protein Homo sapiens 68-71 9787412-11 1998 We did, however, find a prognostic significance with regard to the effect of rejection treatment: in all acute rejections with a CRP < or = 3 (n = 11), steroids were effective. Steroids 155-163 C-reactive protein Homo sapiens 129-132 7677440-10 1995 Erythrocyte sedimentation rate, C reactive protein, and sIL-2R values were significantly less after six months of steroid treatment compared with before treatment. Steroids 114-121 C-reactive protein Homo sapiens 32-50 7579005-7 1995 In five patients with CD in acute phase, investigated before and during the pharmacological treatment (5-aminosalycilic acid and steroids), CRP values fell into the normal range after the second week of therapy, whereas TSA values reached the higher limit of the normal range after the third week, except for two CD patients with a larger location (ileocolonic) of the disease. Steroids 129-137 C-reactive protein Homo sapiens 140-143 8367755-14 1993 After rheumatologic consultation, steroid dosage was titrated to the CRP level and ESR, and vasculitic symptoms resolved. Steroids 34-41 C-reactive protein Homo sapiens 69-72 1403115-11 1992 Also, C-reactive protein determination should be performed and is useful even when steroids are given. Steroids 83-91 C-reactive protein Homo sapiens 6-24 8453797-5 1993 At day 3 and day 5, a significant (p < 0.001) fall in CRP was seen only in the intra-articular steroid group. Steroids 98-105 C-reactive protein Homo sapiens 57-60 2240943-7 1990 The CRP level returned to normal within the first week of steroid therapy for 76% of the patients, before ESR, F and O. Steroids 58-65 C-reactive protein Homo sapiens 4-7 1360960-5 1992 The C-reactive protein (CRP) and erythrocyte sedimentation rate (ESR) improved significantly from 2.55 +/- 0.28(+) and 57.0 +/- 5.69 mm/hr to 0.53 +/- 0.12(+) and 31.2 +/- 3.45 mm/hr, respectively after treatment including steroid and antiplatelet therapy (P < 0.01). Steroids 223-230 C-reactive protein Homo sapiens 4-22 1556702-6 1992 The rate of the correlation between synovial IL-6 level and concentration of serum C-reactive protein in RA was inversely proportional to the dose of steroid treatment in patients with RA. Steroids 150-157 C-reactive protein Homo sapiens 83-101 2065506-1 1991 The effect of intra-articular (i-a) steroid injection on ESR and C-reactive protein (CRP) in rheumatoid arthritis (RA) was investigated. Steroids 36-43 C-reactive protein Homo sapiens 65-83 2065506-1 1991 The effect of intra-articular (i-a) steroid injection on ESR and C-reactive protein (CRP) in rheumatoid arthritis (RA) was investigated. Steroids 36-43 C-reactive protein Homo sapiens 85-88 2280109-12 1990 The adrenal steroids have been continuously administrated and adjusted according to the values of CRP and ESR as well as the cutaneous and mucous manifestations of this disease. Steroids 12-20 C-reactive protein Homo sapiens 98-101 2859021-5 1985 These results indicate that assay of serum CRP provides a precise means of objectively assessing the course of polymyalgia rheumatica during initial therapy with steroids, and suggest that routine measurements of CRP may make a useful contribution to the management of the disease. Steroids 162-170 C-reactive protein Homo sapiens 43-46 34773692-8 2021 The group using the combination of synthetic disease-modifying anti-rheumatic drugs (sDMARD) and steroid therapy had significantly higher HAD-depression, HAD-anxiety, mHCL, DAS28-CRP, HAQ, and MDQ scores than the group using sDMARD alone. Steroids 97-104 C-reactive protein Homo sapiens 179-182 35345571-11 2022 CRP was used by 61% (47/77) of physicians to help identify a secondary infection or determine steroid dose or duration. Steroids 94-101 C-reactive protein Homo sapiens 0-3 34325501-13 2021 The higher initial CRP level in COVID-19 patients might explain the greater steroid responsiveness in this population. Steroids 76-83 C-reactive protein Homo sapiens 19-22 35467748-8 2022 Switching treatment to intensive anti-inflammatory therapy with high-dose steroids and Etanercept finally improved symptom and CRP level associated with the reduction of low absorption areas and disappearance of gas images at bilateral shoulder joints. Steroids 74-82 C-reactive protein Homo sapiens 127-130 35402137-5 2022 Although steroid treatment administered once improved the general condition, her symptoms and C-reactive protein worsened again. Steroids 9-16 C-reactive protein Homo sapiens 94-112 32976826-10 2021 Higher PUCAI score and C-reactive protein on day 3 and 5 of hospital admission predict failure of intravenous steroids. Steroids 110-118 C-reactive protein Homo sapiens 23-41 33355850-0 2021 Can High-Sensitivity C-Reactive Protein Levels Predict Functional Outcome Following Epidural Steroid Injection in Patients with Lumbar Disc Disease? Steroids 93-100 C-reactive protein Homo sapiens 21-39 33367210-3 2020 Steroid use was "biomarker concordant" if given when C-reactive protein (CRP) was >=150 mg/L or withheld when CRP was <150 mg/L, and vice versa for biomarker discordant. Steroids 0-7 C-reactive protein Homo sapiens 53-71 33331351-8 2020 In CD, previous resective surgery, perianal disease and elevated C-reactive protein (CRP) at assessment, while in UC steroid therapy, an elevated CRP and fecal calprotectin at assessment were associated with anemia in a multivariate analysis. Steroids 117-124 C-reactive protein Homo sapiens 85-88 33367210-3 2020 Steroid use was "biomarker concordant" if given when C-reactive protein (CRP) was >=150 mg/L or withheld when CRP was <150 mg/L, and vice versa for biomarker discordant. Steroids 0-7 C-reactive protein Homo sapiens 73-76 33367210-3 2020 Steroid use was "biomarker concordant" if given when C-reactive protein (CRP) was >=150 mg/L or withheld when CRP was <150 mg/L, and vice versa for biomarker discordant. Steroids 0-7 C-reactive protein Homo sapiens 110-113 33205245-9 2021 Patients who were prescribed steroids had significantly higher white cell count (12.4 vs 11.5), CRP (79.5 vs 57) and heart rate (94 vs 88) on admission. Steroids 29-37 C-reactive protein Homo sapiens 96-99 33187976-15 2020 Steroid-sparing treatment strategies may be considered for those IBD patients with higher admission CRP and greater disease duration, who appear to be at the greatest risk of hyperglycaemia. Steroids 0-7 C-reactive protein Homo sapiens 100-103 32012468-6 2020 Once the C-reactive protein value returned to within the normal range after surgery, we reduced the steroid dose. Steroids 100-107 C-reactive protein Homo sapiens 9-27