PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10490838-4	1999	After release from serum deprivation, wild-type and PARP-/-(+PARP) cells, but not PARP-/- cells, exhibited a peak of cells in S phase by 16 h and had progressed through the cell cycle by 22 h. Whereas [3H]thymidine incorporation remained negligible in PARP-/- cells, in vivo DNA replication maximized after 18 h in wild-type and PARP-/-(+PARP) cells.	Thymidine	205-214	poly(ADP-ribose) polymerase 1	Homo sapiens	52-56	
1530662-0	1992	Inhibition of poly(ADP-ribose)polymerase activity by nucleoside analogs of thymidine.	Thymidine	75-84	poly(ADP-ribose) polymerase 1	Homo sapiens	14-40	
8660945-7	1996	Consistently with these results, the addition of PARP inhibitors, such as thymidine and 3-aminobenzamide, during the preincubation with AA, prevents NAD+ depletion and abolishes the protective effect of AA against apoptosis.	Thymidine	74-83	poly(ADP-ribose) polymerase 1	Homo sapiens	49-53	
1530662-2	1992	We evaluated various analogs of deoxythymidine and deoxyuridine as inhibitors of PARP.	Thymidine	32-46	poly(ADP-ribose) polymerase 1	Homo sapiens	81-85	
2968936-2	1988	If the toxins act via this common mechanism, the poly(ADP-ribose) synthetase inhibitors nicotinamide and thymidine would be expected to affect the formation of DNA single-strand breaks in a similar fashion.	Thymidine	105-114	poly(ADP-ribose) polymerase 1	Homo sapiens	49-76	
2968936-5	1988	These results would be expected if nicotinamide and thymidine acted through inhibition of poly(ADP-ribose) synthetase.	Thymidine	52-61	poly(ADP-ribose) polymerase 1	Homo sapiens	90-117