PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9676716-4	1998	In contrast, NAC potentiated both nitroglycerin- and nicorandil-induced vasodilation.	Nitroglycerin	34-47	synuclein alpha	Homo sapiens	13-16	
9676716-6	1998	NAC augmented the nitroglycerin- and nicorandil-induced vasodilation in the small epicardial coronary artery, but not in the large epicardial segments.	Nitroglycerin	18-31	synuclein alpha	Homo sapiens	0-3	
9676716-7	1998	In both groups, NAC potentiated the increase in CBF in response to nitroglycerin.	Nitroglycerin	67-80	synuclein alpha	Homo sapiens	16-19	
10651168-1	1998	We investigated the effects of the sulfhydryl-donor, N-acetylcysteine (NAC), on nitroglycerin (NTG)-induced relaxation of the vascular smooth muscle.	Nitroglycerin	80-93	synuclein alpha	Homo sapiens	71-74	
10651168-1	1998	We investigated the effects of the sulfhydryl-donor, N-acetylcysteine (NAC), on nitroglycerin (NTG)-induced relaxation of the vascular smooth muscle.	Nitroglycerin	95-98	synuclein alpha	Homo sapiens	71-74	
10651168-5	1998	Application of NAC (NTG-NAC) enhanced the relaxing effects of NTG on the histamine-induced tonic contraction rather than the acute contraction.	Nitroglycerin	20-23	synuclein alpha	Homo sapiens	15-18	
10651168-8	1998	These results suggest that treatment with the sulfhydryl donor, NAC, inhibited the phosphorylation of desmin associated with the enhancement of NTG-induced relaxation, which might be related to the mechanisms of recovery from NTG tolerance by sulfhydryl groups.	Nitroglycerin	144-147	synuclein alpha	Homo sapiens	64-67	
10651168-8	1998	These results suggest that treatment with the sulfhydryl donor, NAC, inhibited the phosphorylation of desmin associated with the enhancement of NTG-induced relaxation, which might be related to the mechanisms of recovery from NTG tolerance by sulfhydryl groups.	Nitroglycerin	226-229	synuclein alpha	Homo sapiens	64-67	
8747803-11	1995	Exogenous thiol compounds (e.g. NAC) augments the hypotensive effect of NTG by a tolerance nonspecific mechanism.	Nitroglycerin	72-75	synuclein alpha	Homo sapiens	32-35	
8381319-16	1993	Incubation of GTN (150-1200fJM) for 30min with NAC (0.1-1mM) led to aconcentration-dependent increase in N02- formation.	Nitroglycerin	14-17	synuclein alpha	Homo sapiens	47-50	
8381319-17	1993	The reduced ability of tolerant SMC or EC to potentiate the anti-platelet activity of GTN was restored by NAC (0.5 mM).	Nitroglycerin	86-89	synuclein alpha	Homo sapiens	106-109	
8381319-21	1993	NAC, by directly forming NO from GTN, compensates for this failing mechanism.	Nitroglycerin	33-36	synuclein alpha	Homo sapiens	0-3