PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26334090-6	2015	On the other hand, nitroglycerin-induced relaxation was markedly attenuated by exposing the arteries to H(2)O(2) (pD2: 8.73 +- 0.05 and 8.30 +- 0.05), which was normalized in the presence of catalase (pD2: 8.59 +- 0.05).	Nitroglycerin	19-32	catalase	Rattus norvegicus	191-199	
14732477-10	2004	Nitroglycerin significantly enhanced aortic Mn-SOD, CAT, GR and glutathione-S-transferase (GST) activities and protein expressions with decreased MDA levels, protein carbonyls and BP.	Nitroglycerin	0-13	catalase	Rattus norvegicus	52-55	
12860443-10	2003	Nitroglycerin administration significantly enhanced cardiac Mn-SOD, CAT, and GST activities, and protein expression with decreased MDA levels and BP.	Nitroglycerin	0-13	catalase	Rattus norvegicus	68-71	
12841629-10	2003	Interaction of exercise training and chronic nitroglycerin treatment resulted in normalization of plasma NO, MDA, lactate levels, and CAT activity.	Nitroglycerin	45-58	catalase	Rattus norvegicus	134-137	
8719811-2	1995	In this study we investigated the role of catalase in relaxation induced by hydroxylamine, sodium azide, glyceryl trinitrate and hydrogen peroxide in isolated rings of rat aorta.	Nitroglycerin	105-124	catalase	Rattus norvegicus	42-50	
8719811-14	1995	Pretreatment of endothelium-denuded rings with AT (1-50 mM, 90 min) to inhibit endogenous catalase blocked relaxation induced by sodium azide (1-300 nM) and hydroxylamine (1-300 nM) but had no effect on relaxation induced by hydrogen peroxide (10 microM-1 mM) or glyceryl trinitrate (1-100 nM).	Nitroglycerin	263-282	catalase	Rattus norvegicus	90-98