PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19283361-7	2009	Insulin TET is a third potential site for regulating insulin delivery.	tet	8-11	insulin	Homo sapiens	0-7	
19283361-2	2009	In this review we examine the basis for the hypothesis that insulin acts on the vasculature at three discrete steps to enhance its own delivery to muscle: (1) relaxation of resistance vessels to increase total blood flow; (2) relaxation of pre-capillary arterioles to increase the microvascular exchange surface perfused within skeletal muscle (microvascular recruitment); and (3) the trans-endothelial transport (TET) of insulin.	tet	414-417	insulin	Homo sapiens	60-67	
19283361-9	2009	Recent in vivo and in vitro findings suggest that insulin traverses the vascular endothelium via a trans-cellular, receptor-mediated pathway, and emerging data indicate that insulin acts on the endothelium to facilitate its own TET.	tet	228-231	insulin	Homo sapiens	50-57	
19283361-9	2009	Recent in vivo and in vitro findings suggest that insulin traverses the vascular endothelium via a trans-cellular, receptor-mediated pathway, and emerging data indicate that insulin acts on the endothelium to facilitate its own TET.	tet	228-231	insulin	Homo sapiens	174-181	
11385189-1	2001	However, it remains to be examined how short-term transdermal estrogen therapy (TET) affects insulin sensitivity (SI) in patients with cardiac syndrome X (CSX), who are characterized by elevated insulin resistance.	tet	80-83	insulin	Homo sapiens	93-100	
11872677-6	2002	R(d) was highly correlated with the ISF concentration for insulin and NN304 (r = 0.86 and 0.93, respectively), suggesting that slow transendothelial transport (TET) is responsible for sluggish NN304 action.	tet	160-163	insulin	Homo sapiens	58-65