PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24829152-11	2015	CONCLUSION: Tet preconditioning increased the expression of Bcl-2 and reduced the expression of Caspase-3, thereby attenuating overload training-induced myocardial apoptosis, protecting against overload training-induced myocardial injury, and reducing damage to the myocardium due to overload training.	tet	12-15	BCL2, apoptosis regulator	Rattus norvegicus	60-65	
24829152-8	2015	Conversely, Tet preconditioning significantly increased Bcl-2 expression and the Bcl-2/Bax ratio as compared with the overload training group (P<0.05).	tet	12-15	BCL2, apoptosis regulator	Rattus norvegicus	56-61	
24829152-8	2015	Conversely, Tet preconditioning significantly increased Bcl-2 expression and the Bcl-2/Bax ratio as compared with the overload training group (P<0.05).	tet	12-15	BCL2, apoptosis regulator	Rattus norvegicus	81-86	
25434925-8	2015	The immunohistochemical, RT-PCR, and western blot analyses indicated that Tet treatment significantly increased the Bcl-2+ brain cells and the mRNA and protein expression levels of Bcl-2, and decreased the Bax+ cells and the mRNA and protein expression levels of Bax, as well as elevated the ratio of Bcl-2/Bax, in phenobarbital-dependent and -withdrawn rats.	tet	74-77	BCL2, apoptosis regulator	Rattus norvegicus	116-121	
25434925-8	2015	The immunohistochemical, RT-PCR, and western blot analyses indicated that Tet treatment significantly increased the Bcl-2+ brain cells and the mRNA and protein expression levels of Bcl-2, and decreased the Bax+ cells and the mRNA and protein expression levels of Bax, as well as elevated the ratio of Bcl-2/Bax, in phenobarbital-dependent and -withdrawn rats.	tet	74-77	BCL2, apoptosis regulator	Rattus norvegicus	181-186	
25434925-8	2015	The immunohistochemical, RT-PCR, and western blot analyses indicated that Tet treatment significantly increased the Bcl-2+ brain cells and the mRNA and protein expression levels of Bcl-2, and decreased the Bax+ cells and the mRNA and protein expression levels of Bax, as well as elevated the ratio of Bcl-2/Bax, in phenobarbital-dependent and -withdrawn rats.	tet	74-77	BCL2, apoptosis regulator	Rattus norvegicus	181-186	
25434925-10	2015	Tet alleviates the phenobarbital withdrawal symptoms and protects the brain cells against apoptosis, which may be a result of the regulation of the mRNA and protein expression levels of Bcl-2 and Bax.	tet	0-3	BCL2, apoptosis regulator	Rattus norvegicus	186-191