PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19135070-7	2009	The protective effect of TNF-alpha was similar in magnitude to the protective effects of NMDA and DETA-NONOate, both of which are NO-cGMP-PKG pathway activators.	2,2'-(hydroxynitrosohydrazono)bis-ethanamine	98-110	protein kinase cGMP-dependent 1	Homo sapiens	138-141	
24977346-6	2014	Incubation with DETA NONOate (a stable NO donor) and 8-Br-cGMP (a cell membrane permeable analog of cGMP) attenuated Akt phosphorylation at Ser-473, which was prevented by Rp-8-Br-PET-cGMPS (a specific inhibitor of PKG) and calyculin A (an inhibitor of protein phosphatase 1 and 2A) but not by okadaic acid (a selective inhibitor of protein phosphatase 2A).	2,2'-(hydroxynitrosohydrazono)bis-ethanamine	16-28	protein kinase cGMP-dependent 1	Homo sapiens	215-218	
17891157-3	2007	EXPERIMENTAL APPROACH: Effects of a PKG inhibitor on the basal tension and responses induced by nitroglycerin, DETA NONOate, and 8-Br-cGMP in isolated porcine coronary veins were determined.	2,2'-(hydroxynitrosohydrazono)bis-ethanamine	111-123	protein kinase cGMP-dependent 1	Homo sapiens	36-39	
17891157-8	2007	Relaxation of the veins induced by nitroglycerin and DETA NONOate was accompanied with increases in cGMP content and PKG activity.	2,2'-(hydroxynitrosohydrazono)bis-ethanamine	53-65	protein kinase cGMP-dependent 1	Homo sapiens	117-120	
17377806-7	2007	The activity of PKG but not that of PKA was increased by nitroglycerin and DETA NONOate in intact vessels and increased by cGMP in the tissue homogenates.	2,2'-(hydroxynitrosohydrazono)bis-ethanamine	75-87	protein kinase cGMP-dependent 1	Homo sapiens	16-19