PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 28586070-7 2017 The results demonstrated that the protective effect of diethylcarbamazine inhibited isoproterenol-induced AMI through the suppression of inflammation, iNOS, TGF-beta1, COX-2 and the PARP pathway, and revealed the clinical potential of diethylcarbamazine for therapeutic and clinical applications. Diethylcarbamazine 55-73 transforming growth factor, beta 1 Rattus norvegicus 157-166