PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28586070-7	2017	The results demonstrated that the protective effect of diethylcarbamazine inhibited isoproterenol-induced AMI through the suppression of inflammation, iNOS, TGF-beta1, COX-2 and the PARP pathway, and revealed the clinical potential of diethylcarbamazine for therapeutic and clinical applications.	Diethylcarbamazine	55-73	transforming growth factor, beta 1	Rattus norvegicus	157-166