PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7549418-6	1995	Injection of VIP or SP reduced methacholine-induced sweating to a similar degree in all groups, except that the reduction was smaller in the non-neuropathic group than in the others (p = 0.028 versus normal subjects, p = 0.014 versus neuropathic diabetic patients).	Methacholine Chloride	31-43	vasoactive intestinal peptide	Homo sapiens	13-16	
1671333-4	1991	Both basal and methacholine (M.chol)-induced MLGP secretion could be blocked by VIP (1 pM to 1 microM) and by isoproterenol (ISO) (0.1 nM to 10 nM) in a concentration-dependent and reversible manner.	Methacholine Chloride	15-27	vasoactive intestinal peptide	Homo sapiens	80-83	
8348252-0	1993	Responses of plasma vasoactive intestinal polypeptide to methacholine and exercise loading in children and adolescents with bronchial asthma.	Methacholine Chloride	57-69	vasoactive intestinal peptide	Homo sapiens	20-53	
8348252-1	1993	Responses of plasma vasoactive intestinal polypeptide (VIP) to methacholine inhalation and to exercise loading were studied in asthmatic patients to clarify a significant role of the peptide.	Methacholine Chloride	63-75	vasoactive intestinal peptide	Homo sapiens	20-53	
8348252-1	1993	Responses of plasma vasoactive intestinal polypeptide (VIP) to methacholine inhalation and to exercise loading were studied in asthmatic patients to clarify a significant role of the peptide.	Methacholine Chloride	63-75	vasoactive intestinal peptide	Homo sapiens	55-58	
3895438-2	1985	At a dose of 0.03 to 0.12 nanomole, VIP produced a dose-dependent, prolonged (3 to 15 minutes) depolarization of the ganglion and enhanced the ganglionic depolarization elicited by the muscarinic agonist acetyl-beta-methylcholine.	Methacholine Chloride	204-229	vasoactive intestinal peptide	Homo sapiens	36-39	
2851006-3	1988	VIP at a low concentration (10(-9) M), which did not produce any significant increases over controls, produced a 2.4- to 5-fold augmentation of the glycoconjugate release induced by 10(-9) to 10(-7) M methacholine (MCh).	Methacholine Chloride	201-213	vasoactive intestinal peptide	Homo sapiens	0-3	
2851006-3	1988	VIP at a low concentration (10(-9) M), which did not produce any significant increases over controls, produced a 2.4- to 5-fold augmentation of the glycoconjugate release induced by 10(-9) to 10(-7) M methacholine (MCh).	Methacholine Chloride	215-218	vasoactive intestinal peptide	Homo sapiens	0-3	
2827508-9	1987	The VIP-induced cAMP level was markedly augmented by MCh and further enhanced by Iso with or without theophylline.	Methacholine Chloride	53-56	vasoactive intestinal peptide	Homo sapiens	4-7	
3891019-4	1985	The intra-arterial administration of VIP (1-50 micrograms/kg) enhanced the postganglionic discharge elicited by the muscarinic agonist, acetyl-beta-methylcholine, but did not alter the postganglionic firing elicited by the nicotinic agonist, tetramethylammonium or by electrical stimulation of preganglionic axons in the pelvic nerve.	Methacholine Chloride	136-161	vasoactive intestinal peptide	Homo sapiens	37-40	
3891019-7	1985	VIP suppressed the muscarinic inhibition of ganglionic transmission produced by acetyl-beta-methylcholine without altering the response to other inhibitory agents (norepinephrine, leucine-enkephalin and gamma-aminobutyric acid (GABA).	Methacholine Chloride	80-105	vasoactive intestinal peptide	Homo sapiens	0-3	
6152040-2	1984	The membrane depolarization evoked by the muscarinic agonist, acetyl-beta-methylcholine in these neurons was similarly enhanced by VIP.	Methacholine Chloride	62-87	vasoactive intestinal peptide	Homo sapiens	131-134	
7305106-3	1981	In explants of 9 mucosal specimens of normal airways, VIP (10 ng to 1 micrograms/ml) caused a dose-dependent inhibition of baseline and methacholine-stimulated release of both glycoconjugates and lysozyme.	Methacholine Chloride	136-148	vasoactive intestinal peptide	Homo sapiens	54-57	
7305106-4	1981	At a concentration (1 micrograms/ml) that caused maximal inhibition of glycoconjugate and lysozyme release, VIP also caused a small inhibition of baseline but not methacholine-induced discharge of labeled macromolecules from mucous and serous cells of the submucosal glands.	Methacholine Chloride	163-175	vasoactive intestinal peptide	Homo sapiens	108-111	
7305106-6	1981	By contrast, VIP did inhibit baseline and methacholine-stimulated release of lysozyme, but this was less marked than in explants of normal airways.	Methacholine Chloride	42-54	vasoactive intestinal peptide	Homo sapiens	13-16