PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22092211-9	2011	A mitoK(ATP) channels inhibitor, 5-hydroxydecanoic acid (200 muM) and an opener, diazoxide (100 muM), were used to investigate the involvement of mitoK(ATP) channels.	Diazoxide	81-90	latexin	Homo sapiens	96-99	
21145327-4	2011	In single-channel recordings, cell exposure to diazoxide (30 muM) significantly prolonged the mean open time of K(ATP) channels; however, neither closed-time kinetics nor the single-channel conductance of the channel was altered by this compound.	Diazoxide	47-56	latexin	Homo sapiens	61-64	
17503968-6	2007	Higher diazoxide concentrations (>/=30 muM) decreased glucagon and insulin secretion, and alpha- and beta-cell Ca(2+) responses, in parallel.	Diazoxide	7-16	latexin	Homo sapiens	42-45	
29130561-3	2018	The stimulatory effect of the compound (10 muM) on glucose uptake on adipose tissue was blocked by diazoxide, wortmannin, U73122, colchicine, and N-ethylmaleimide.	Diazoxide	99-108	latexin	Homo sapiens	43-46	
27753906-8	2016	Diazoxide (300 muM), cromakalim (10 muM) nicorandil (10 muM) which is known to activate KATP channel also completely blocked vasomotion in a glibenclamide sensitive manner.	Diazoxide	0-9	latexin	Homo sapiens	15-18	
26205024-11	2015	EPO (10 IU/ml) and diazoxide (100 muM) treatments significantly increased (p <0.01) whereas glibenclamide decreased ( p<0.05) HIF-1 alpha mRNA expression.	Diazoxide	19-28	latexin	Homo sapiens	34-37