PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12791941-5	2003	RESULTS: The mitoK(ATP) channel opener diazoxide (100 micromol/L) significantly suppressed the number of cells with terminal deoxynucleotidyl transferase-mediated dUTP nick end-labeling (TUNEL)-positive nuclei and the increase in caspase-3 activity induced by 20 micromol/L H2O2.	Diazoxide	39-48	caspase 3	Homo sapiens	230-239	
31912704-4	2019	Both diazoxide and diphenyleneiodonium increased cell viability by inhibiting the increase in reactive oxygen species and caspase-3 activity as well as the decrease in Bcl-2 induced by amyloid beta.	Diazoxide	5-14	caspase 3	Homo sapiens	122-131	
26205024-6	2015	Evaluation of the dose and time dependent effects of EPO, glibenclamide and diazoxide on apoptosis was done by caspase-3 activity levels.	Diazoxide	76-85	caspase 3	Homo sapiens	111-120	
20398465-9	2010	CONCLUSION: The diazoxide may reduce the expression of caspase-3, intracellular translocation of Omi/HtrA2, and stability of mitochondrial membrane potential, thereby significantly alleviates HK-2 cells injury induced by post-asphyxial-serum of neonate.	Diazoxide	16-25	caspase 3	Homo sapiens	55-64