PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32336187-2	2020	Recently a case of congenital hyperinsulinemic hypoglycemia and a severe neurodevelopmental syndrome due to a mutation in the voltage-gated Cav1.3 Ca2+ channel CACNA1D gene has been reported which required long-term treatment with diazoxide.	Diazoxide	231-240	calcium voltage-gated channel subunit alpha1 D	Homo sapiens	160-167	
32336187-5	2020	The patient was treated with diazoxide and nifedipine with adequate control of glucose metabolism and blood pressure, and with improvement in muscle tone.Our findings further confirm the pathogenic role of CACNA1D for congentital hyperinsulinemic hypoglycemia and primary aldosteronism.	Diazoxide	29-38	calcium voltage-gated channel subunit alpha1 D	Homo sapiens	206-213