PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26423578-0	2015	Po2 cycling protects diaphragm function during reoxygenation via ROS, Akt, ERK, and mitochondrial channels.	PO-2	0-3	thymoma viral proto-oncogene 1	Mus musculus	70-73	
26423578-4	2015	We hypothesize that Po2 cycling effectively increases muscle fatigue resistance through reactive oxygen species (ROS), protein kinase B (Akt), extracellular signal-regulated kinase (ERK), and certain mitochondrial channels during reoxygenation.	PO-2	20-23	thymoma viral proto-oncogene 1	Mus musculus	137-140	
26423578-10	2015	Thus we have identified a unique signaling pathway that may involve ROS, Akt, ERK, and mitochondrial channels responsible for Po2 cycling protection during reoxygenation conditions in the diaphragm.	PO-2	126-129	thymoma viral proto-oncogene 1	Mus musculus	73-76	
28774345-8	2017	In addition, analysis of the liver specimens indicated that injection with the 1% pO2 secretome maximized the expression of the intermediate molecules of the PIP3/Akt and IL-6/STAT3 signaling pathways, all of which are known to promote liver regeneration.	PO-2	82-85	thymoma viral proto-oncogene 1	Mus musculus	163-166