PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25724126-4	2015	RESULTS: Our stratified analysis based on the value of the conventional TIN parameter N-acetylglucosaminidase (NAG) excretion showed that the high-NAG index group (more than median value of NAG-to-Cr ratio, n = 47) demonstrated significantly high FEMg values (p = 0.017).	Chromium	197-199	N-acetyl-alpha-glucosaminidase	Homo sapiens	111-114	
25724126-4	2015	RESULTS: Our stratified analysis based on the value of the conventional TIN parameter N-acetylglucosaminidase (NAG) excretion showed that the high-NAG index group (more than median value of NAG-to-Cr ratio, n = 47) demonstrated significantly high FEMg values (p = 0.017).	Chromium	197-199	N-acetyl-alpha-glucosaminidase	Homo sapiens	147-150	
25724126-4	2015	RESULTS: Our stratified analysis based on the value of the conventional TIN parameter N-acetylglucosaminidase (NAG) excretion showed that the high-NAG index group (more than median value of NAG-to-Cr ratio, n = 47) demonstrated significantly high FEMg values (p = 0.017).	Chromium	197-199	N-acetyl-alpha-glucosaminidase	Homo sapiens	147-150	
33713715-8	2021	With respect to Cd and to some extent Cr, beta-2-microglobulin (B2-MG) and N-acetyl-beta-D-glucosaminidase (NAG) are well-established, sensitive, and the most common effect biomarkers to relate Cd or Cr exposure to renal tubular dysfunction.	Chromium	200-202	N-acetyl-alpha-glucosaminidase	Homo sapiens	108-111	
30238383-11	2019	There were significant associations between elevated glu/Cr and asphyxia or neonatal acute kidney injury, and elevated NAG/Cr and indomethacin use, although these were not confirmed by multivariate analysis.	Chromium	123-125	N-acetyl-alpha-glucosaminidase	Homo sapiens	119-122	
30238383-12	2019	At follow-up, the frequency of elevated NAG/Cr, glu/Cr, UA/Cr, and malb/Cr was reduced but that of elevated Ca/Cr, IgG/Cr, and beta2m/Cr remained similar or increased.	Chromium	44-46	N-acetyl-alpha-glucosaminidase	Homo sapiens	40-43	
26980004-3	2016	Of the 20 participants in this study, the urinary NAG/creatinine (Cr) rose from a pre-exercise level of 8.9, 6.7 to 14.3 (mumol/L/mmol Cr; median, IQR) to 12.9, 9.1 to 17.7 post exercise (p = 0.0003, Mann Whitney U test).	Chromium	66-68	N-acetyl-alpha-glucosaminidase	Homo sapiens	50-53	
26980004-3	2016	Of the 20 participants in this study, the urinary NAG/creatinine (Cr) rose from a pre-exercise level of 8.9, 6.7 to 14.3 (mumol/L/mmol Cr; median, IQR) to 12.9, 9.1 to 17.7 post exercise (p = 0.0003, Mann Whitney U test).	Chromium	135-137	N-acetyl-alpha-glucosaminidase	Homo sapiens	50-53	
26980004-5	2016	Participants with a rise in the NAG/Cr ratio post exercise had a greater ability to increase endothelial reactivity (%; median, IQR; 2.56, 0.1 to 3.7) cf.	Chromium	36-38	N-acetyl-alpha-glucosaminidase	Homo sapiens	32-35	
26980004-9	2016	The mechanism by which the phenotypically distinct sub-group of patients with intermittent claudication who experience a NAG/Cr rise involves complex interactions between systemic oxidative stress and endothelial function.	Chromium	125-127	N-acetyl-alpha-glucosaminidase	Homo sapiens	121-124	
9852385-5	1999	The urinary NAG/Cr ratio is influenced by fasting, parity, gestation and age.	Chromium	16-18	N-acetyl-alpha-glucosaminidase	Homo sapiens	12-15	
33592134-17	2021	Urinary NAG-CR, but not ACR, increased during follow-up.	Chromium	12-14	N-acetyl-alpha-glucosaminidase	Homo sapiens	8-11