PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8996639-5	1996	Other ionic flux inhibitors 4,4"-diisothiocyanostilbene-2,2"-disulfonic acid (DIDS) and furosemide slightly inhibited the pHi recovery from an acid load.	Furosemide	88-98	glucose-6-phosphate isomerase	Rattus norvegicus	122-125	
7515562-4	1994	Na(+)-H+ antiport activity, estimated from the Na(+)-induced initial rate of pHi recovery of Na(+)-depleted acidified cells in the presence of 0.1 mM furosemide to inhibit Na(+)-K(+)-2Cl- cotransport, was inhibited by 300 mM urea and 10(-8) M arginine vasopressin (AVP) in an additive manner.	Furosemide	150-160	glucose-6-phosphate isomerase	Rattus norvegicus	77-80	
7810602-6	1994	An NMDG-Cl- dependent NH4(+)-induced fall in pHi was reduced approximately 33% by 10 mM Ba+, approximately 84% by 0.1 mM bumetanide, and 100% by 1.5 mM furosemide, whereas 1 mM hydrochlorothiazide had no effect; inhibition by Ba+ was observed even in the presence of 0.1 mM verapamil added to block both K+ channels and K+/NH4+ antiport.	Furosemide	152-162	glucose-6-phosphate isomerase	Rattus norvegicus	45-48	
8035168-6	1994	Luminal furosemide (10(-4) M) reduced the initial rate of cell acidification by 70% and the fall in steady state pHi by 35%.	Furosemide	8-18	glucose-6-phosphate isomerase	Rattus norvegicus	113-116	
8035168-10	1994	Addition of furosemide to the lumen abolished net ammonium absorption and caused pHi to increase abruptly (dpHi/dt = 0.8 U/min) to 7.0.	Furosemide	12-22	glucose-6-phosphate isomerase	Rattus norvegicus	81-84