PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1823389-0	1991	Evaluation of insulin-induced changes in the renal response to furosemide in normal subjects.	Furosemide	63-73	insulin	Homo sapiens	14-21	
26438710-10	2015	This hypothesis was confirmed by a video recording that revealed the administration by the mother of multiple drugs (insulin, glyburide, progesterone, and furosemide) that mimicked most of the features of Rabson-Mendenhall syndrome, including hirsutism and hypoglycemia with coincident, inappropriately normal c-peptide values due to the administration of the insulin secretagogue.	Furosemide	155-165	insulin	Homo sapiens	310-319	
26438710-10	2015	This hypothesis was confirmed by a video recording that revealed the administration by the mother of multiple drugs (insulin, glyburide, progesterone, and furosemide) that mimicked most of the features of Rabson-Mendenhall syndrome, including hirsutism and hypoglycemia with coincident, inappropriately normal c-peptide values due to the administration of the insulin secretagogue.	Furosemide	155-165	insulin	Homo sapiens	360-367	
20413337-12	2010	CONCLUSION: Furosemide is physically compatible with bicarbonate solution, heparin, insulin, morphine and nitroglycerin and incompatible with amiodarone, cisatracurium, haloperidol, midazolam and urapidil.	Furosemide	12-22	insulin	Homo sapiens	84-91	
11125222-8	2000	Likewise, a reduction of insulin-induced swelling by the loop diuretics furosemide and bumetanide cause insulin resistance shown by the levels of cell swelling, MAP-kinase activation and proteolysis control.	Furosemide	72-82	insulin	Homo sapiens	25-32	
14985360-9	2004	Inhibition of insulin-induced swelling by furosemide largely abolished activation of beta(1) integrin and phosphorylation of Src, but not of PKB.	Furosemide	42-52	insulin	Homo sapiens	14-21	
1823389-2	1991	We analyzed the response of the kidney to furosemide in 5 healthy men, in the presence of both normal physiological serum insulin levels and levels at the upper limit of the physiological range, obtained by the hyperinsulinemic-euglycemic clamp technique.	Furosemide	42-52	insulin	Homo sapiens	122-129	
1823389-3	1991	After furosemide administration, glomerular filtration rate, urine flow, urine sodium excretion, free water clearance, urine pH, plasma renin activity and plasma aldosterone exhibited the same behavior in the presence of both serum insulin concentrations.	Furosemide	6-16	insulin	Homo sapiens	232-239	
1823389-4	1991	The rise in urinary potassium excretion following furosemide administration was significantly lower in the presence of high insulin concentrations.	Furosemide	50-60	insulin	Homo sapiens	124-131	
1823389-6	1991	Thus, in conditions in which natriuresis is mildly stimulated, as in the case of the administration of low doses of furosemide, insulin does not affect the rate of renal sodium reabsorption.	Furosemide	116-126	insulin	Homo sapiens	128-135	
2203383-2	1990	It is possible that these factors may play a role in changes in insulin sensitivity in vivo produced by such diverse conditions as treatment with furosemide, thyroid status or catecholamine status.	Furosemide	146-156	insulin	Homo sapiens	64-71	
2083062-1	1990	The study has been performed in an attempt to provide further data on the supposed direct action of insulin on the kidney, based on the assumption that any effect of insulin on sodium reabsorption via co-transport should shift the dose-response curve to the furosemide administration.	Furosemide	258-268	insulin	Homo sapiens	100-107	
2083062-1	1990	The study has been performed in an attempt to provide further data on the supposed direct action of insulin on the kidney, based on the assumption that any effect of insulin on sodium reabsorption via co-transport should shift the dose-response curve to the furosemide administration.	Furosemide	258-268	insulin	Homo sapiens	166-173	
7013722-8	1980	It is concluded that the stimulation of insulin and glucagon secretion observed in vitro with high furosemide concentrations (5 mmol/l) are not observed under usual therapeutic conditions.	Furosemide	99-109	insulin	Homo sapiens	40-47	
3073092-3	1988	So, for instance, drugs that increase prostaglandins synthesis as colchicine or furosemide inhibit insulin secretion while non steroid anti-inflammator drugs, mainly salicylates, which inhibit cyclo-oxygenase, enhance the insulin response to various stimuli.	Furosemide	80-90	insulin	Homo sapiens	99-106	
6998800-0	1980	Effect of furosemide on insulin and glucagon responses to arginine in normal subjects.	Furosemide	10-20	insulin	Homo sapiens	24-31	
6998800-1	1980	This study aimed at evaluating the influence of furosemide upon insulin and glucagon responses to arginine in healthy subjects.	Furosemide	48-58	insulin	Homo sapiens	64-71	
6998800-3	1980	The acute insulin response (mean change from 3-10 min) to the second arginine pulse was significantly inhibited by furosemide (mean increase: 14.8 +/- 3.0 microU/ml versus 11.7 +/- 2.5 microU/ml, p < 0.01).	Furosemide	115-125	insulin	Homo sapiens	10-17	
6998800-7	1980	The effect of furosemide on insulin and glucagon secretion might be mediated through enhanced release of endogenous prostaglandin E.	Furosemide	14-24	insulin	Homo sapiens	28-35	
467810-0	1979	Acetylsalicyclic acid restores acute insulin response reduced by furosemide in man.	Furosemide	65-75	insulin	Homo sapiens	37-44	
467810-6	1979	Infusion of lysine acetylsalicylate (LAS), an inhibitor of endogenous PGE synthesis, completely reversed the inhibitory effect of furosemide on insulin secretion and also augmented acute insulin response to glucose (response before furosemide + LAS: 41 +/- 6 muU/ml; during furosemide + LAS: 50 +/- 7 muU/ml, N = 10, P less than 0.02).	Furosemide	130-140	insulin	Homo sapiens	144-151	
467810-8	1979	These findings demonstrate that (1) furosemide inhibits glucose-induced acute insulin responses and (2) LAS completely reverses the inhibitory effect of furosemide and also accelerates glucose disposal.	Furosemide	36-46	insulin	Homo sapiens	78-85	
467810-9	1979	It is suggested that furosemide acts via the release of endogenous PGEs, which are known to inhibit insulin responses in man.	Furosemide	21-31	insulin	Homo sapiens	100-107	
4996929-0	1970	[Glucagon test and electrolytes in long term administration of furosemide to insulin requiring diabetics].	Furosemide	63-73	insulin	Homo sapiens	77-84