PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22946656-6	2012	Exposure to caspase inhibitors, Fas-antagonistic antibody, DR4 antagonist, and furosemide (a blocker of Bax translocation) effectively abolished ar-turmerone-triggered apoptosis.	Furosemide	79-89	BCL2 associated X, apoptosis regulator	Homo sapiens	104-107	
17172466-4	2007	Inhibition of Bax translocation using furosemide, as well as Bax gene deletion, afforded significant protection against malonate-induced apoptosis.	Furosemide	38-48	BCL2 associated X, apoptosis regulator	Homo sapiens	14-17	
18546202-2	2008	The chloride channel inhibitor furosemide prevented the intracellular acidification, the translocation of Bax and the cell death.	Furosemide	31-41	BCL2 associated X, apoptosis regulator	Homo sapiens	106-109	
11864976-5	2002	The increase in the content of Bax was followed by the translocation of this protein from the cytosol to the mitochondria, an event that was inhibited by furosemide, a chloride channel inhibitor.	Furosemide	154-164	BCL2 associated X, apoptosis regulator	Homo sapiens	31-34	
12393866-3	2002	The chloride channel inhibitor furosemide prevented intracellular alkalinization, Bax translocation, cytochrome c release, and cell death.	Furosemide	31-41	BCL2 associated X, apoptosis regulator	Homo sapiens	82-85	
12393866-13	2002	It is concluded that with either staurosporine or TNF a furosemide-sensitive change in pH(i) is linked to Bax translocation, cytochrome c release, and cell killing.	Furosemide	56-66	BCL2 associated X, apoptosis regulator	Homo sapiens	106-109