PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27917784-3	2016	METHODS: We targeted the acetyl-lysine binding modules or bromodomains, which are associated with c-MYC transcriptional activation.	N(alpha)-acetyllysine	25-38	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	98-103	
21889194-2	2011	Toward this objective, we have targeted MYC transcription by interfering with chromatin-dependent signal transduction to RNA polymerase, specifically by inhibiting the acetyl-lysine recognition domains (bromodomains) of putative coactivator proteins implicated in transcriptional initiation and elongation.	N(alpha)-acetyllysine	168-181	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	40-43	
34563497-8	2021	In recent years, inhibiting the interaction of acetyl-lysine residues and bromodomain proteins on chromatin has served as an interesting target to regulate the expression of various pathological genes, including BCL-2, MYC, and NF-kappaB.	N(alpha)-acetyllysine	47-60	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	219-222