PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6594681-1	1984	Differential enzymic analyses of the erythrocyte glutamic-oxaloacetic transaminase and the erythrocyte glutathione reductase of a patient with a 3-yr history of the carpal tunnel syndrome (CTS) revealed high deficiencies of both vitamin B-6 and riboflavin as based on approximately equal to 30% levels of the specific activities of these enzymes.	Vitamin B 6	229-240	transthyretin	Homo sapiens	189-192	
8733640-1	1996	Case reports and small case series suggest that vitamin B6 deficiency is an important etiologic factor in carpal tunnel syndrome (CTS).	Vitamin B 6	48-58	transthyretin	Homo sapiens	130-133	
24312849-3	2013	The present study set out to assess the effect of vitamin B6 in patients with CTS.	Vitamin B 6	50-60	transthyretin	Homo sapiens	78-81	
24312849-10	2013	CONCLUSION: The present study suggests that vitamin B6 treatment improves clinical symptoms and sensory electrodiagnostic results in CTS patients, and thus is recommended for CTS treatment.	Vitamin B 6	44-54	transthyretin	Homo sapiens	133-136	
24312849-10	2013	CONCLUSION: The present study suggests that vitamin B6 treatment improves clinical symptoms and sensory electrodiagnostic results in CTS patients, and thus is recommended for CTS treatment.	Vitamin B 6	44-54	transthyretin	Homo sapiens	175-178	
8733640-8	1996	Furthermore, in our opinion, empiric prescription of vitamin B6 to patients with CTS is unwarranted and potentially hazardous.	Vitamin B 6	53-63	transthyretin	Homo sapiens	81-84