PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 7557098-1 1995 BACKGROUND & AIMS: It has been reported that patients with chronic renal failure have low serum aspartate aminotransferase (AST) and alanine aminotransferase (ALT) levels as a result of vitamin B6 deficiency. Vitamin B 6 190-200 solute carrier family 17 member 5 Homo sapiens 100-126 7557098-1 1995 BACKGROUND & AIMS: It has been reported that patients with chronic renal failure have low serum aspartate aminotransferase (AST) and alanine aminotransferase (ALT) levels as a result of vitamin B6 deficiency. Vitamin B 6 190-200 solute carrier family 17 member 5 Homo sapiens 128-131 7557098-4 1995 METHODS: Serum levels of vitamin B6 and its coenzyme were reassessed in relation to AST and ALT levels in patients undergoing long-term hemodialysis using high-performance liquid chromatography. Vitamin B 6 25-35 solute carrier family 17 member 5 Homo sapiens 84-87 3175340-5 1988 The present study may come to the following conclusions: 1) The relative activation rate of plasma AST-m activity by PLP may be a reliable index of vitamin B6 nutritional status during pregnancy. Vitamin B 6 148-158 solute carrier family 17 member 5 Homo sapiens 99-102 2737364-7 1989 Vitamin B6 nutriture, as determined by erythrocyte aspartate aminotransferase (AST) and alanine aminotransferase (ALT) activities, was normal in all diabetic subjects before vitamin B6 therapy. Vitamin B 6 0-10 solute carrier family 17 member 5 Homo sapiens 51-77 2737364-7 1989 Vitamin B6 nutriture, as determined by erythrocyte aspartate aminotransferase (AST) and alanine aminotransferase (ALT) activities, was normal in all diabetic subjects before vitamin B6 therapy. Vitamin B 6 0-10 solute carrier family 17 member 5 Homo sapiens 79-82 7026088-1 1981 Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P). Vitamin B 6 168-178 solute carrier family 17 member 5 Homo sapiens 0-26 7026088-1 1981 Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P). Vitamin B 6 168-178 solute carrier family 17 member 5 Homo sapiens 28-31 7026088-1 1981 Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P). Vitamin B 6 168-178 solute carrier family 17 member 5 Homo sapiens 222-225 675385-3 1978 The enzyme AST requires pyridoxal-5-phosphate (PLP) (active vitamin B6) as a co-enzyme to express its activity. Vitamin B 6 60-70 solute carrier family 17 member 5 Homo sapiens 11-14 675385-4 1978 Since approximately 90% of patients with severe cirrhosis are vitamin B6-deficient, it has been suggested that vitamin B6 supplements given to these patients might cause an elevation of falsely low serum AST concentrations. Vitamin B 6 111-121 solute carrier family 17 member 5 Homo sapiens 204-207 3008634-1 1986 In vitro supplementation with the active form of vitamin B6, pyridoxal-phosphate (PLP), increases measurements of both serum aminotransferase enzymes, L-aspartate: 2-oxoglutarate amino transferase, EC 2.6.1.1 (AST) and L-alanine: 2-oxoglutarate aminotransferase, EC 2.6.1.2 (ALT). Vitamin B 6 49-59 solute carrier family 17 member 5 Homo sapiens 210-213