PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7557098-1	1995	BACKGROUND & AIMS: It has been reported that patients with chronic renal failure have low serum aspartate aminotransferase (AST) and alanine aminotransferase (ALT) levels as a result of vitamin B6 deficiency.	Vitamin B 6	190-200	solute carrier family 17 member 5	Homo sapiens	100-126	
7557098-1	1995	BACKGROUND & AIMS: It has been reported that patients with chronic renal failure have low serum aspartate aminotransferase (AST) and alanine aminotransferase (ALT) levels as a result of vitamin B6 deficiency.	Vitamin B 6	190-200	solute carrier family 17 member 5	Homo sapiens	128-131	
7557098-4	1995	METHODS: Serum levels of vitamin B6 and its coenzyme were reassessed in relation to AST and ALT levels in patients undergoing long-term hemodialysis using high-performance liquid chromatography.	Vitamin B 6	25-35	solute carrier family 17 member 5	Homo sapiens	84-87	
3175340-5	1988	The present study may come to the following conclusions: 1) The relative activation rate of plasma AST-m activity by PLP may be a reliable index of vitamin B6 nutritional status during pregnancy.	Vitamin B 6	148-158	solute carrier family 17 member 5	Homo sapiens	99-102	
2737364-7	1989	Vitamin B6 nutriture, as determined by erythrocyte aspartate aminotransferase (AST) and alanine aminotransferase (ALT) activities, was normal in all diabetic subjects before vitamin B6 therapy.	Vitamin B 6	0-10	solute carrier family 17 member 5	Homo sapiens	51-77	
2737364-7	1989	Vitamin B6 nutriture, as determined by erythrocyte aspartate aminotransferase (AST) and alanine aminotransferase (ALT) activities, was normal in all diabetic subjects before vitamin B6 therapy.	Vitamin B 6	0-10	solute carrier family 17 member 5	Homo sapiens	79-82	
7026088-1	1981	Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P).	Vitamin B 6	168-178	solute carrier family 17 member 5	Homo sapiens	0-26	
7026088-1	1981	Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P).	Vitamin B 6	168-178	solute carrier family 17 member 5	Homo sapiens	28-31	
7026088-1	1981	Aspartate aminotransferase (AST) determinations in erythrocytes of patients undergoing chronic haemodialysis and with kidney transplants showed that patients receiving vitamin B6 had a smaller relative stimulation rate of AST by pyridoxal-5"-phosphate (P-5-P).	Vitamin B 6	168-178	solute carrier family 17 member 5	Homo sapiens	222-225	
675385-3	1978	The enzyme AST requires pyridoxal-5-phosphate (PLP) (active vitamin B6) as a co-enzyme to express its activity.	Vitamin B 6	60-70	solute carrier family 17 member 5	Homo sapiens	11-14	
675385-4	1978	Since approximately 90% of patients with severe cirrhosis are vitamin B6-deficient, it has been suggested that vitamin B6 supplements given to these patients might cause an elevation of falsely low serum AST concentrations.	Vitamin B 6	111-121	solute carrier family 17 member 5	Homo sapiens	204-207	
3008634-1	1986	In vitro supplementation with the active form of vitamin B6, pyridoxal-phosphate (PLP), increases measurements of both serum aminotransferase enzymes, L-aspartate: 2-oxoglutarate amino transferase, EC 2.6.1.1 (AST) and L-alanine: 2-oxoglutarate aminotransferase, EC 2.6.1.2 (ALT).	Vitamin B 6	49-59	solute carrier family 17 member 5	Homo sapiens	210-213