PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21882541-7	2011	Supplementation of vitamin B6 to cases with the genetic defect of GLO1 before onset of psychosis is suggested to be possible strategy for prevention of schizophrenia until pubertal stage since such mutation carriers could have been exposed by high level of AGEs for a long time before disease onset.	Vitamin B 6	19-29	glyoxalase I	Homo sapiens	66-70	
24646262-2	2014	We first identified a patient with a mutation in GLO1 (glyoxalase I) who showed increased AGE (advanced glycation end-product) levels and decreased vitamin B6 levels.	Vitamin B 6	148-158	glyoxalase I	Homo sapiens	49-53	
24646262-2	2014	We first identified a patient with a mutation in GLO1 (glyoxalase I) who showed increased AGE (advanced glycation end-product) levels and decreased vitamin B6 levels.	Vitamin B 6	148-158	glyoxalase I	Homo sapiens	55-67	
34198071-4	2021	Moreover, we found that vitamin B6 (VB6) levels in peripheral blood of the schizophrenia patients with GLO1 dysfunction are significantly lower than that of healthy controls.	Vitamin B 6	24-34	glyoxalase I	Homo sapiens	103-107	
34198071-4	2021	Moreover, we found that vitamin B6 (VB6) levels in peripheral blood of the schizophrenia patients with GLO1 dysfunction are significantly lower than that of healthy controls.	Vitamin B 6	36-39	glyoxalase I	Homo sapiens	103-107