PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 22931421-8 2013 AZT down-regulated the pro-proliferative genes encoding AKT1, MYC, STAT1, MAPK8, MAPK9, CCL-3, Bcl-3, and cyclin D2; pro-angiogenenic genes encoding VEGF and IL8; and genes involved in cell adhesion (ICAM1 and FN1) and the NF-kappaB pathway. Zidovudine 0-3 MYC proto-oncogene, bHLH transcription factor Homo sapiens 62-65 16132531-9 2005 Of the telomerase subunits, telomerase reverse transcriptase (hTERT) and c-Myc were the first to show a reduction in activity after AZT treatment, followed by changes in hTER , Mad1 and hTEP-1. Zidovudine 132-135 MYC proto-oncogene, bHLH transcription factor Homo sapiens 73-78 16132531-10 2005 CONCLUSION: Cyclic treatment with AZT initially suppressed hTERT and c-Myc, followed by suppression of hTER, Mad1 and hTEP-1. Zidovudine 34-37 MYC proto-oncogene, bHLH transcription factor Homo sapiens 69-74 27633795-8 2016 Regarding extratelomeric activities, our results showed a decrease of 64, 38 and 25% in the transcription of c-Myc, Cyc-D1 and TERT, respectively (p<0.05) after AZT treatment. Zidovudine 164-167 MYC proto-oncogene, bHLH transcription factor Homo sapiens 109-114 26680867-9 2002 Among the telomerase subunits, hTERT and c-Myc were the first factors to change after AZT treatment, subsequently, followed by the changes of hTR, Mad1 and TEP. Zidovudine 86-89 MYC proto-oncogene, bHLH transcription factor Homo sapiens 41-46 26680867-10 2002 CONCLUSION: The suppression of hTERT and c-Myc by AZT treatment was the initial genetic phenomenon, subsequently followed by the changes of hTR, Mad1 and TEP. Zidovudine 50-53 MYC proto-oncogene, bHLH transcription factor Homo sapiens 41-46