PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19398959-10	2009	AZT worsens cardiac dysfunction and increases mitochondrial H(2)O(2) in SOD2(+/-) KO.	Zidovudine	0-3	superoxide dismutase 2, mitochondrial	Mus musculus	72-76	
19398959-11	2009	Conversely, both SOD2-OX and mCAT TGs prevent or attenuate AZT-induced cardiac oxidative stress and LV dysfunction.	Zidovudine	59-62	superoxide dismutase 2, mitochondrial	Mus musculus	17-21	
25260898-0	2014	Overexpression of mitochondrial antioxidant manganese superoxide dismutase (MnSOD) provides protection against AZT- or 3TC-induced endothelial dysfunction.	Zidovudine	111-114	superoxide dismutase 2, mitochondrial	Mus musculus	44-74	
25260898-0	2014	Overexpression of mitochondrial antioxidant manganese superoxide dismutase (MnSOD) provides protection against AZT- or 3TC-induced endothelial dysfunction.	Zidovudine	111-114	superoxide dismutase 2, mitochondrial	Mus musculus	76-81	
9224727-6	1997	Other parameters associated with loss of Mn-SOD such as increase in carbonyl proteins and decrease of sulfhydryl content were also significantly enhanced by AZT in Tat mice.	Zidovudine	157-160	superoxide dismutase 2, mitochondrial	Mus musculus	41-47	
35631648-7	2022	Zidovudine/lamivudine-treated mice had elevated malondialdehyde levels (oxidative stress marker) compared to other groups and lower mRNA levels of manganese superoxide dismutase and peroxisome proliferator-activated receptor gamma coactivator 1-alpha in the placenta compared to tenofovir/emtricitabine-treated mice.	Zidovudine	0-10	superoxide dismutase 2, mitochondrial	Mus musculus	147-177