PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30471476-6	2019	Moreover, the use of ethanol conditioning in conjunction with dynamic culture induces pro-vascularization bioactivity of EC-derived EVs that is correlated with increased EV levels of pro-angiogenic lncRNAs HOTAIR and MALAT1.	Ethanol	21-28	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	217-223	
18353916-8	2008	The results suggest that a selective pharmacological remedy could thus be devised against the rewarding effects of ethanol that are postulated to mediate alcohol abuse and addiction, targeting the specific HCN genes expressed in dopaminergic neurons.	Ethanol	115-122	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	206-209	
16708609-6	2006	It was also shown that the increased ethanol caused a drop in HbCO and HCN levels.	Ethanol	37-44	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	71-74	
22560368-4	2012	Comparison of mRNA levels of MALAT-1 was performed by northern and dot blot experiments using different regions of brain from human alcoholics and rats chronically treated with ethanol vapours and following withdrawal.	Ethanol	177-184	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	29-36	
29062004-6	2017	Further, ethanol-induced upregulation of long non-coding RNAs (lncRNAs) HOTAIR and MALAT1 in endothelial EVs was observed to play a significant role in mediating pro-angiogenic effects of these vesicles.	Ethanol	9-16	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	83-89