PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21441352-10	2011	In addition, M1 protein-induced Mac-1 expression on neutrophils was abolished by simvastatin.	Simvastatin	81-92	integrin subunit alpha M	Homo sapiens	32-37	
23250325-0	2013	Simvastatin abrogates inflamed neutrophil adhesive properties, in association with the inhibition of Mac-1 integrin expression and modulation of Rho kinase activity.	Simvastatin	0-11	integrin subunit alpha M	Homo sapiens	101-106	
23250325-5	2013	Neutrophil surface expression of the Mac-1 integrin subunit, CD11b, was augmented by TNF-alpha, and this increased expression was also inhibited by simvastatin.	Simvastatin	148-159	integrin subunit alpha M	Homo sapiens	37-42	
23250325-5	2013	Neutrophil surface expression of the Mac-1 integrin subunit, CD11b, was augmented by TNF-alpha, and this increased expression was also inhibited by simvastatin.	Simvastatin	148-159	integrin subunit alpha M	Homo sapiens	61-66	
9350917-10	1997	Treatment of these patients with the HMG-CoA reductase inhibitors lovastatin or simvastatin (20 to 40 mg/day) for 6 weeks slightly decreased total and LDL cholesterol plasma levels and monocyte CD11b surface expression but resulted in a significant reduction of monocyte adhesion to endothelium (p < 0.01, n = 7).	Simvastatin	80-91	integrin subunit alpha M	Homo sapiens	194-199	
19125317-7	2009	After simvastatin treatment, CD11b expression decreased to 116.9 +/- 12.5 (P < 0.03) and CD14 expression to 107.5 +/- 6.2 (P < 0.04).	Simvastatin	6-17	integrin subunit alpha M	Homo sapiens	29-34	
12695460-10	2003	However, both neutrophil CD11b expression and endothelial P-selectin exocytosis were less in the simvastatin groups than in the controls.	Simvastatin	97-108	integrin subunit alpha M	Homo sapiens	25-30