PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25625122-10	2015	The drop in PR expression together with altered distribution of p21 and p27 can explain different effects of cyclin E isoforms expression on progesterone responsivity.	Progesterone	141-153	H3 histone pseudogene 16	Homo sapiens	64-67	
30145226-1	2019	We previously demonstrated that progesterone (P4) up-regulated p53 expression, which in turn increased p21 and p27 expression, and finally resulted in proliferation inhibition in human umbilical vein endothelial cells (HUVEC).	Progesterone	32-44	H3 histone pseudogene 16	Homo sapiens	103-106	
18850315-4	2008	The formation of CDK2-p21 and CDK2-p27 were increased and the CDK2 activity was decreased in the progesterone-treated HUVEC.	Progesterone	97-109	H3 histone pseudogene 16	Homo sapiens	22-25	
24401087-12	2014	Moreover, Stat6 overexpression and knockdown, respectively, increased or prevented the induction of p21 and p27 gene expression by progesterone.	Progesterone	131-143	H3 histone pseudogene 16	Homo sapiens	100-103	
18850315-6	2008	Transfection of HUVEC with dominant negative p53 cDNA prevented the progesterone-induced increases in p21 and p27 promoter activity and protein level, decreases in thymidine incorporation, and capillary-like tube formation.	Progesterone	68-80	H3 histone pseudogene 16	Homo sapiens	102-105	
16550965-7	2006	CONCLUSION: p21 and p27 may be related to progesterone-induced growth suppression in human endometrial adenocarcinoma.	Progesterone	42-54	H3 histone pseudogene 16	Homo sapiens	12-15	
15520195-12	2004	The morphologic change and p21 up-regulation still were observed after Pg treatment.	Progesterone	71-73	H3 histone pseudogene 16	Homo sapiens	27-30	
9328342-10	1997	Instead, a second progesterone dose delays the fall of p21 and enhances the rise of p27(Kip1), thereby intensifying the progesterone resistance in an autoinhibitory loop.	Progesterone	18-30	H3 histone pseudogene 16	Homo sapiens	55-58