PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 15615861-11 2005 Therefore, our data suggest that the NGFI-B may play a significant role in up-regulation of HSD3B2 that leads to the increase in progesterone production that is seen in granulosa cells at ovulation. Progesterone 129-141 nuclear receptor subfamily 4 group A member 1 Homo sapiens 37-43 15767916-12 2005 Hormone therapy with progestogens (hormone receptor expression of the tumor is 71% for estrogens and 95% for progesterone) is widely studied in the literature with a 46% response rate and 46% rate of disease stabilization. Progesterone 109-121 nuclear receptor subfamily 4 group A member 1 Homo sapiens 35-51 11719525-2 2002 We have shown that Nur77 plays an important role in ovarian physiology by mediating the PGF(2alpha) induction of 20alpha-HSD, a steroidogenic enzyme involved in the catabolism of progesterone. Progesterone 179-191 nuclear receptor subfamily 4 group A member 1 Homo sapiens 19-24 1655735-2 1991 The overproduced hMR binds aldosterone with high affinity (Kd = 1.36 nM) and has high affinity for cortisol, cortexolone, and progesterone. Progesterone 126-138 nuclear receptor subfamily 4 group A member 1 Homo sapiens 17-20 34383180-5 2021 Hormone receptor positivity was defined as estrogen receptor-positive, progesterone-positive and HER 2-negative cancer. Progesterone 71-83 nuclear receptor subfamily 4 group A member 1 Homo sapiens 0-16 34773204-6 2022 Through decidualization induction of the human endometrial stromal cells (hESCs) cultured in vitro and additional P treatment, the results of chromatin immunoprecipitation and other experiments show that the P treatment could upregulate the expression of NR4A1 in hESCs, and this process was mediated under the direct effect of progesterone receptor (PR) and NR4A1. Progesterone 114-115 nuclear receptor subfamily 4 group A member 1 Homo sapiens 255-260 34773204-6 2022 Through decidualization induction of the human endometrial stromal cells (hESCs) cultured in vitro and additional P treatment, the results of chromatin immunoprecipitation and other experiments show that the P treatment could upregulate the expression of NR4A1 in hESCs, and this process was mediated under the direct effect of progesterone receptor (PR) and NR4A1. Progesterone 114-115 nuclear receptor subfamily 4 group A member 1 Homo sapiens 359-364 34773204-6 2022 Through decidualization induction of the human endometrial stromal cells (hESCs) cultured in vitro and additional P treatment, the results of chromatin immunoprecipitation and other experiments show that the P treatment could upregulate the expression of NR4A1 in hESCs, and this process was mediated under the direct effect of progesterone receptor (PR) and NR4A1. Progesterone 208-209 nuclear receptor subfamily 4 group A member 1 Homo sapiens 255-260 34773204-6 2022 Through decidualization induction of the human endometrial stromal cells (hESCs) cultured in vitro and additional P treatment, the results of chromatin immunoprecipitation and other experiments show that the P treatment could upregulate the expression of NR4A1 in hESCs, and this process was mediated under the direct effect of progesterone receptor (PR) and NR4A1. Progesterone 208-209 nuclear receptor subfamily 4 group A member 1 Homo sapiens 359-364 34773204-7 2022 When the NR4A1 in hESCs was silenced, the promotion of hESC proliferation by P was inhibited. Progesterone 77-78 nuclear receptor subfamily 4 group A member 1 Homo sapiens 9-14 220946-0 1978 Hormone receptor levels and metabolic activity in the uterus of the ewe: regulation by oestradiol and progesterone. Progesterone 102-114 nuclear receptor subfamily 4 group A member 1 Homo sapiens 0-16 6721511-3 1984 At dichophase , prior to the onset of the S-stage in cell division cycle, under conjugation of progesterone with a certain specific type of protein (hormone receptor) the cells are induced toward the development of secretory function, i.e. toward differentiation, resulting in no synthesis of DNA. Progesterone 95-107 nuclear receptor subfamily 4 group A member 1 Homo sapiens 149-165 33863896-1 2021 Expression of hormone receptor (HR) for estrogens (ER) and progesterone (PR) and HER2 remains the cornerstone to define the therapeutic strategy for breast cancer patients. Progesterone 59-71 nuclear receptor subfamily 4 group A member 1 Homo sapiens 14-30 196828-0 1977 Control of hormone receptor levels and growth of 7,12-dimethylbenz(a)anthracene-induced mammary tumors by estrogens, progesterone and prolactin. Progesterone 117-129 nuclear receptor subfamily 4 group A member 1 Homo sapiens 11-27 33863896-1 2021 Expression of hormone receptor (HR) for estrogens (ER) and progesterone (PR) and HER2 remains the cornerstone to define the therapeutic strategy for breast cancer patients. Progesterone 59-71 nuclear receptor subfamily 4 group A member 1 Homo sapiens 32-34 33495599-0 2021 CYP3A7*1C allele: linking premenopausal oestrone and progesterone levels with risk of hormone receptor-positive breast cancers. Progesterone 53-65 nuclear receptor subfamily 4 group A member 1 Homo sapiens 86-102 27388585-10 2016 PIK3CA, PTEN, and AKT1 mutations occurred more frequently in the presence of hormone receptor overexpression (androgen, progesterone, or estrogen receptor). Progesterone 120-132 nuclear receptor subfamily 4 group A member 1 Homo sapiens 77-93 24485460-4 2014 Integration of an endothelial genome-wide transcriptional profile with chromatin immunoprecipitation sequencing revealed that PR induces an NR4A1 (Nur77/TR3)-dependent transcriptional program that broadly regulates vascular permeability in response to progesterone. Progesterone 252-264 nuclear receptor subfamily 4 group A member 1 Homo sapiens 140-145 24485460-4 2014 Integration of an endothelial genome-wide transcriptional profile with chromatin immunoprecipitation sequencing revealed that PR induces an NR4A1 (Nur77/TR3)-dependent transcriptional program that broadly regulates vascular permeability in response to progesterone. Progesterone 252-264 nuclear receptor subfamily 4 group A member 1 Homo sapiens 147-152 24485460-4 2014 Integration of an endothelial genome-wide transcriptional profile with chromatin immunoprecipitation sequencing revealed that PR induces an NR4A1 (Nur77/TR3)-dependent transcriptional program that broadly regulates vascular permeability in response to progesterone. Progesterone 252-264 nuclear receptor subfamily 4 group A member 1 Homo sapiens 153-156