PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 29764928-11 2018 Furthermore, our in vivo results also showed that GRP78, CHOP expression and apoptosis were significantly increased in endometrial cells during the secretory phase as well as by in vitro treatment with progesterone. Progesterone 202-214 DNA damage inducible transcript 3 Homo sapiens 57-61 31718641-13 2019 CONCLUSIONS: MPA may activate ER stress by progesterone-PRB pathway to up-regulate CHOP expression, which may be one of the molecular mechanisms underlying the inhibitory effect of MPA on EC cells with PRB+. Progesterone 43-55 DNA damage inducible transcript 3 Homo sapiens 83-87 29239061-0 2018 Progesterone inhibited endoplasmic reticulum stress associated apoptosis induced by interleukin-1beta via the GRP78/PERK/CHOP pathway in BeWo cells. Progesterone 0-12 DNA damage inducible transcript 3 Homo sapiens 121-125 29239061-10 2018 IL-1beta could induce ERS and associated cell apoptosis by activating the GRP78/PERK/CHOP signal pathway, which could be inhibited by progesterone. Progesterone 134-146 DNA damage inducible transcript 3 Homo sapiens 85-89 12082616-6 2002 GADD153 expression was upregulated by the lactogenic hormones insulin and progesterone and associated with differentiation of normal mammary epithelial cells. Progesterone 74-86 DNA damage inducible transcript 3 Homo sapiens 0-7