PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31718641-13	2019	CONCLUSIONS: MPA may activate ER stress by progesterone-PRB pathway to up-regulate CHOP expression, which may be one of the molecular mechanisms underlying the inhibitory effect of MPA on EC cells with PRB+.	Progesterone	43-55	DNA damage inducible transcript 3	Homo sapiens	83-87	
12082616-6	2002	GADD153 expression was upregulated by the lactogenic hormones insulin and progesterone and associated with differentiation of normal mammary epithelial cells.	Progesterone	74-86	DNA damage inducible transcript 3	Homo sapiens	0-7	
29239061-0	2018	Progesterone inhibited endoplasmic reticulum stress associated apoptosis induced by interleukin-1beta via the GRP78/PERK/CHOP pathway in BeWo cells.	Progesterone	0-12	DNA damage inducible transcript 3	Homo sapiens	121-125	
29239061-10	2018	IL-1beta could induce ERS and associated cell apoptosis by activating the GRP78/PERK/CHOP signal pathway, which could be inhibited by progesterone.	Progesterone	134-146	DNA damage inducible transcript 3	Homo sapiens	85-89	
29764928-11	2018	Furthermore, our in vivo results also showed that GRP78, CHOP expression and apoptosis were significantly increased in endometrial cells during the secretory phase as well as by in vitro treatment with progesterone.	Progesterone	202-214	DNA damage inducible transcript 3	Homo sapiens	57-61