PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9590261-6	1998	Thus, progesterone is a potent inhibitor of steady state levels TNF-alpha mRNA and TNF-alpha protein production in activated macrophages and may achieve this result through effects on an inhibitor of NF-kappaB.	Progesterone	6-18	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	200-209	
25080336-10	2014	Progesterone suppressed LPS-induced NF-kappaB activation by decreasing inhibitory kappaBalpha and NF-kappaB p65 phosphorylation and p65 nuclear translocation.	Progesterone	0-12	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	36-45	
25080336-10	2014	Progesterone suppressed LPS-induced NF-kappaB activation by decreasing inhibitory kappaBalpha and NF-kappaB p65 phosphorylation and p65 nuclear translocation.	Progesterone	0-12	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	98-107	
27500900-0	2016	Endometrial breakdown with sustained progesterone release involves NF-kappaB-mediated functional progesterone withdrawal in a mouse implant model.	Progesterone	37-49	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	67-76	
27500900-5	2016	Using co-immunoprecipitation assays, we observed the constitutive activation of nuclear factor kappa-b (NF-kappaB) p65 and its interaction with the progesterone receptor (PGR); moreover, transcriptional activity of the PGR was also repressed by NF-kappaB activity in primary mouse and human decidual stromal cells that mimic progesterone maintenance.	Progesterone	148-160	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	80-102	
27500900-5	2016	Using co-immunoprecipitation assays, we observed the constitutive activation of nuclear factor kappa-b (NF-kappaB) p65 and its interaction with the progesterone receptor (PGR); moreover, transcriptional activity of the PGR was also repressed by NF-kappaB activity in primary mouse and human decidual stromal cells that mimic progesterone maintenance.	Progesterone	148-160	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	104-113	
27500900-5	2016	Using co-immunoprecipitation assays, we observed the constitutive activation of nuclear factor kappa-b (NF-kappaB) p65 and its interaction with the progesterone receptor (PGR); moreover, transcriptional activity of the PGR was also repressed by NF-kappaB activity in primary mouse and human decidual stromal cells that mimic progesterone maintenance.	Progesterone	148-160	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	245-254	
27500900-7	2016	In vivo comparison of endometrial breakdown induced by progesterone withdrawal to that seen during sustained progesterone exposure, in the presence of NF-kappaB inhibitors, revealed that NF-kappaB-mediated functional progesterone withdrawal is involved in endometrial breakdown in this implant model.	Progesterone	55-67	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	187-196	
27500900-7	2016	In vivo comparison of endometrial breakdown induced by progesterone withdrawal to that seen during sustained progesterone exposure, in the presence of NF-kappaB inhibitors, revealed that NF-kappaB-mediated functional progesterone withdrawal is involved in endometrial breakdown in this implant model.	Progesterone	109-121	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	187-196