PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16135805-6	2005	Bigenic PRAI-SRC-1(-/-) mice revealed that SRC-1 modulates PR activity in the uterus in a cell-specific fashion and is involved in PR gene activation in stroma and myometrium of the uterus in response to estrogen and progesterone.	Progesterone	217-229	nuclear receptor coactivator 1	Mus musculus	43-48	
26098207-3	2015	WT dams crossed with males deficient for both SRC-1 and SRC-2 had suppressed myometrial inflammation, increased serum progesterone, and delayed parturition, which could be reconciled by injection of either SP-A or PAF into the amnion.	Progesterone	118-130	nuclear receptor coactivator 1	Mus musculus	46-51	
16141356-3	2006	In the mammary gland, PR activity in the luminal epithelium of both wild-type and SRC-1(-/-) mice was induced by estrogen + progesterone treatment.	Progesterone	124-136	nuclear receptor coactivator 1	Mus musculus	82-87	
16135805-7	2005	In contrast, SRC-1 was involved in the down-regulation of PR target gene expression in the luminal and glandular epithelial compartments of the uterus after chronic progesterone treatment.	Progesterone	165-177	nuclear receptor coactivator 1	Mus musculus	13-18	
12021388-8	2002	However, overexpression of SRC-1 counteracted progesterone to repress mCAR activity.	Progesterone	46-58	nuclear receptor coactivator 1	Mus musculus	27-32